PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19926935-8	2009	The apoptosis by xanthorrhizol was markedly evidenced by induction of DNA fragmentation, release of cytochrome c, activation of caspases, and cleavage of poly-(ADP-ribose) polymerase.	xanthorrhizol	17-30	cytochrome c, somatic	Homo sapiens	100-112	
19189649-11	2008	These data suggest treatment with xanthorrhizol modulates MDA-MB-231 cell apoptosis through the mitochondria-mediated pathway subsequent to the disruption of mitochondrial transmembrane potential, release of cytochrome c, activation of caspase-3 and caspase-9, and the modulation of PARP-1 protein.	xanthorrhizol	34-47	cytochrome c, somatic	Homo sapiens	208-220	
34664399-6	2022	In addition, XNT treatment alters the DeltaPsim, thereby induces apoptosis was closely coordinated with the induction of pro-apoptotic markers Bax, Bad, caspase- 3, 9 and cytochrome c, and suppression of anti-apoptotic (Bcl-2, Bcl-XL) protein expression.	xanthorrhizol	13-16	cytochrome c, somatic	Homo sapiens	171-183