PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 17724165-8 2007 The addition of phosphatidic acid, a reaction product of PLD, partially restored fMLP-mediated transepithelial migration in the presence of 1-butanol but not the migration of Mac-1-deficient neutrophil-differentiated HL-60 cells. 1-Butanol 140-149 formyl peptide receptor 1 Homo sapiens 81-85 17724165-7 2007 Incubation in 1-butanol did inhibit fMLP but not C5a-mediated migration across intestinal epithelial cell monolayers, showing that transepithelial migration to fMLP but not C5a is dependent on PLD. 1-Butanol 14-23 formyl peptide receptor 1 Homo sapiens 36-40 17724165-7 2007 Incubation in 1-butanol did inhibit fMLP but not C5a-mediated migration across intestinal epithelial cell monolayers, showing that transepithelial migration to fMLP but not C5a is dependent on PLD. 1-Butanol 14-23 formyl peptide receptor 1 Homo sapiens 160-164 11427526-3 2001 Western blot analysis using phosphospecific p38 and p44/p42 mitogen-activated protein kinase antibodies showed that increasing concentrations of ethanol or 1-butanol but not 2-butanol (0.05-0.5%) inhibited fMLP-induced p38 activation but did not inhibit p44/p42 activation. 1-Butanol 156-165 formyl peptide receptor 1 Homo sapiens 206-210 15294123-6 2004 In 1-butanol+LPS+fMLP group, PLD activity of preoperative neutrophils was lower than that in LPS+fMLP group (P<0.01), besides the release of elastase and MPO decreased sharply below both LPS+fMLP and fMLP groups (P<0.01). 1-Butanol 3-12 formyl peptide receptor 1 Homo sapiens 17-21 15294123-6 2004 In 1-butanol+LPS+fMLP group, PLD activity of preoperative neutrophils was lower than that in LPS+fMLP group (P<0.01), besides the release of elastase and MPO decreased sharply below both LPS+fMLP and fMLP groups (P<0.01). 1-Butanol 3-12 formyl peptide receptor 1 Homo sapiens 97-101 15294123-6 2004 In 1-butanol+LPS+fMLP group, PLD activity of preoperative neutrophils was lower than that in LPS+fMLP group (P<0.01), besides the release of elastase and MPO decreased sharply below both LPS+fMLP and fMLP groups (P<0.01). 1-Butanol 3-12 formyl peptide receptor 1 Homo sapiens 97-101 15294123-6 2004 In 1-butanol+LPS+fMLP group, PLD activity of preoperative neutrophils was lower than that in LPS+fMLP group (P<0.01), besides the release of elastase and MPO decreased sharply below both LPS+fMLP and fMLP groups (P<0.01). 1-Butanol 3-12 formyl peptide receptor 1 Homo sapiens 97-101 15294123-11 2004 Notably, PLD activity was even nonstatistically lower in 1-butanol+LPS+fMLP group than that in LPS or fMLP group. 1-Butanol 57-66 formyl peptide receptor 1 Homo sapiens 71-75 10931844-8 2000 PLD inhibitors 1-butanol and 2, 3-diphosphoglycerate, or the ARF6(N48R) mutant assumed to be defective in PLD activation, blocked fMLP-elicited oxidase activity in transfected cells. 1-Butanol 15-24 formyl peptide receptor 1 Homo sapiens 130-134 10986013-10 2000 In addition, the TMCH- and fMLP-induced DCF fluorescence, after the preincubation with the phospholipase D modulator n-butanol, was lowered by 83 and 52%, respectively. 1-Butanol 117-126 formyl peptide receptor 1 Homo sapiens 27-31 10860542-9 2000 However, fMLP-mediated AA release was reduced by approximately 45% by Clostridium difficile toxin B (10 ng/ml) or by 1-butanol; both block phospholipase D (PLD) activity. 1-Butanol 117-126 formyl peptide receptor 1 Homo sapiens 9-13