PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10215671-5	1999	Propranolol completely reversed the rise in heart rate and cardiac output, the fall in peripheral resistance, the shortening of electromechanical systole, and the rise in insulin; it blunted the increase in free fatty acids and gastrin.	Propranolol	0-11	gastrin	Homo sapiens	228-235	
6377465-2	1984	dl-Propranolol inhibited the gastrin response to hypoglycaemia markedly and more than the insignificant alteration observed after d-propranolol.	Propranolol	0-14	gastrin	Homo sapiens	29-36	
2216936-1	1990	The purpose of the study was to evaluate the effect of 6 weeks treatment with hydrochlorothiazide or propranolol on gastric acid and gastrin secretion in essential hypertension.	Propranolol	101-112	gastrin	Homo sapiens	133-140	
2694115-0	1989	[Effect of propranolol therapy on the secretion of insulin, glucagon, gastrin and pancreatic polypeptide in patients with essential hypertension].	Propranolol	11-22	gastrin	Homo sapiens	70-77	
1672696-4	1991	The results demonstrated that epinephrine and terbutaline (a beta 2 agonist) significantly stimulated gastrin release above basal which could be blocked by the addition of propranolol (beta-adrenergic antagonist).	Propranolol	172-183	gastrin	Homo sapiens	102-109	
976301-7	1976	Isoproterenol stimulated gastrin secretion was completely suppressed by propranolol, a beta-adrenergic receptor blocking agent, which had no effect on meal stimulated gastrin secretion.	Propranolol	72-83	gastrin	Homo sapiens	25-32	
6120877-4	1982	Propranolol reduced distention-induced gastrin release by approximately 90% (p less than 0.02), whereas phentolamine had no significant effect on the gastrin response to distention.	Propranolol	0-11	gastrin	Homo sapiens	39-46	
6120877-5	1982	In additional experiments, we evaluated the effect of the same doses of propranolol or phentolamine on the exaggerated gastrin response to gastric distention that occurred during cholinergic blockade with atropine.	Propranolol	72-83	gastrin	Homo sapiens	119-126	
6120877-7	1982	), propranolol significantly (p less than 0.01) reduced distention-induced gastrin release, whereas phentolamine significantly enhanced the gastrin response to distention (p less than 0.01).	Propranolol	3-14	gastrin	Homo sapiens	75-82	
6120877-8	1982	We conclude that: (1) distention-induced gastrin release was reduced by propranolol, suggesting that gastric distention releases gastrin by a beta-adrenergic mechanism and (2) distention-induced gastrin release was enhanced by phentolamine, but only in the presence of atropine.	Propranolol	72-83	gastrin	Homo sapiens	41-48	
6120877-8	1982	We conclude that: (1) distention-induced gastrin release was reduced by propranolol, suggesting that gastric distention releases gastrin by a beta-adrenergic mechanism and (2) distention-induced gastrin release was enhanced by phentolamine, but only in the presence of atropine.	Propranolol	72-83	gastrin	Homo sapiens	129-136	
6120877-8	1982	We conclude that: (1) distention-induced gastrin release was reduced by propranolol, suggesting that gastric distention releases gastrin by a beta-adrenergic mechanism and (2) distention-induced gastrin release was enhanced by phentolamine, but only in the presence of atropine.	Propranolol	72-83	gastrin	Homo sapiens	129-136	
6104918-4	1980	The increase in plasma gastrin induced by arginine pulse was significantly suppressed by the infusion of beta-adrenergic blocking agent, propranolol, while the infusion of alpha-adrenergic blocking agent, phentolamine tended to enhance the arginine-induced gastrin secretion slightly but not significantly.	Propranolol	137-148	gastrin	Homo sapiens	23-30	
379428-0	1979	[Effect of propranolol on the gastrin concentration in the serum and on hydrochloric acid secretion in duodenal peptic ulcer].	Propranolol	11-22	gastrin	Homo sapiens	30-37	
964476-1	1976	The influence of propranolol, 50 mug/kg administered intravenously, on the gastrin concentration during sham feeding and after a test meal was studied in eight normal subjects.	Propranolol	17-28	gastrin	Homo sapiens	75-82	
1064146-4	1976	Propranolol eliminated the gastrin response to hypoglycaemia, pindolol reduced it, and practolol caused no alteration.	Propranolol	0-11	gastrin	Homo sapiens	27-34	
241651-0	1975	The effect of propranolol and phentolamine on serum gastrin concentration in response to respiratory acidosis in normal man.	Propranolol	14-25	gastrin	Homo sapiens	52-59	
241651-5	1975	50 mug/kg propranolol intravenously produced a decrease of gastrin concentrations from 145 to 127 pg/ml (p less than 0.01) and a total suppression of hypergastrinaemia in response to CO2 rebreathing, suggesting activation of beta-cell receptors in respiratory acidosis.	Propranolol	10-21	gastrin	Homo sapiens	59-66	
1205007-1	1975	In ten normal subjects the effect of propranolol on serum gastrin concentration and HC1 secretion during insulin hypoglycemia (0.2 U/kg) was studied.	Propranolol	37-48	gastrin	Homo sapiens	58-65	
1205007-2	1975	Under the influence of propranolol (50 mug/kg intravenously in 3 min) the gastrin response to insulin was abolished.	Propranolol	23-34	gastrin	Homo sapiens	74-81