PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18854141-3	2008	When activity of the Target of Rapamycin (TOR), a core component of the nutrient-responsive pathway, is reduced in the PG, the ecdysone peak that marks the end of larval development is abrogated.	Ecdysone	127-135	Target of rapamycin	Drosophila melanogaster	21-40	
18854141-3	2008	When activity of the Target of Rapamycin (TOR), a core component of the nutrient-responsive pathway, is reduced in the PG, the ecdysone peak that marks the end of larval development is abrogated.	Ecdysone	127-135	Target of rapamycin	Drosophila melanogaster	42-45	
18854141-7	2008	In conclusion, the PG uses TOR signaling to couple nutritional input with ecdysone production and developmental timing.	Ecdysone	74-82	Target of rapamycin	Drosophila melanogaster	27-30	
25421296-3	2014	Previous studies indicate that insulin/insulin-like growth factor signaling (IIS)/Target of Rapamycin (TOR) signaling in the prothoracic glands (PGs) regulates ecdysone biosynthesis and critical weight.	Ecdysone	160-168	Target of rapamycin	Drosophila melanogaster	82-101	
25421296-3	2014	Previous studies indicate that insulin/insulin-like growth factor signaling (IIS)/Target of Rapamycin (TOR) signaling in the prothoracic glands (PGs) regulates ecdysone biosynthesis and critical weight.	Ecdysone	160-168	Target of rapamycin	Drosophila melanogaster	103-106	
22654869-4	2012	Furthermore, they have shown that the insulin/TOR pathway interacts with hormonal systems, like ecdysone and juvenile hormone, to regulate the timing of development and hence the duration of growth.	Ecdysone	96-104	Target of rapamycin	Drosophila melanogaster	46-49