PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 34390101-0 2021 Cyanides, Isocyanides, and Hydrides of Zn, Cd and Hg from Metal Atom and HCN Reactions: Matrix Infrared Spectra and Electronic Structure Calculations. Cadmium 43-45 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 73-76 34770852-1 2021 Carbonate MCO3 (M = Zn, Cd) can act as both Lewis acid and base to engage in a spodium bond with nitrogen-containing bases (HCN, NHCH2, and NH3) and a chalcogen bond with SeHX (X = F, Cl, OH, OCH3, NH2, and NHCH3), respectively. Cadmium 24-26 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 124-127 34390101-1 2021 Zinc and cadmium atoms from laser ablation of the metals and mercury atoms ablated from a dental amalgam target react with HCN in excess argon during deposition at 5 K to form the MCN and MNC molecules and CN radicals. Cadmium 9-16 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 123-126 34897851-1 2022 An investigation of pulsed-laser-ablated Zn, Cd and Hg metal atom reactions with HCN under excess argon during co-deposition with laser-ablated Hg atoms from a dental amalgam target also provided Hg emissions capable of photoionization of the CN photo-dissociation product. Cadmium 45-47 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 81-84 30090505-0 2017 LncRNA-MALAT1 as a novel biomarker of cadmium toxicity regulates cell proliferation and apoptosis. Cadmium 38-45 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 7-13 34106509-1 2021 MCO 3 (M=Zn, Cd, Hg) engages in a spodium bond with nitrogen-containing bases (HCN, NHCH 2 , NH 3 ) and a pnicogen bond with FH 2 Z (Z=P, As, Sb). Cadmium 13-15 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 79-82 30090505-2 2017 This study aimed to investigate whether lncRNA-MALAT1 could serve as a novel biomarker of Cd toxicity in cells, animals and Cd-exposed workers, and regulate cell proliferation, apoptosis, migration and invasion. Cadmium 90-92 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 47-53 30090505-2 2017 This study aimed to investigate whether lncRNA-MALAT1 could serve as a novel biomarker of Cd toxicity in cells, animals and Cd-exposed workers, and regulate cell proliferation, apoptosis, migration and invasion. Cadmium 124-126 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 47-53 30090505-5 2017 Cadmium increased MALAT1 expression in the lung of Cd-exposed rats in a dose-dependent manner. Cadmium 0-7 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 18-24 30090505-5 2017 Cadmium increased MALAT1 expression in the lung of Cd-exposed rats in a dose-dependent manner. Cadmium 51-53 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 18-24 30090505-6 2017 A significant positive correlation was observed between blood MALAT1 expression and urinary/blood Cd concentrations, and there were significant correlations of MALAT1 expression with the expressions of target genes in the lung of Cd-exposed rats and the blood of Cd exposed workers. Cadmium 98-100 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 62-68 30090505-6 2017 A significant positive correlation was observed between blood MALAT1 expression and urinary/blood Cd concentrations, and there were significant correlations of MALAT1 expression with the expressions of target genes in the lung of Cd-exposed rats and the blood of Cd exposed workers. Cadmium 230-232 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 62-68 30090505-6 2017 A significant positive correlation was observed between blood MALAT1 expression and urinary/blood Cd concentrations, and there were significant correlations of MALAT1 expression with the expressions of target genes in the lung of Cd-exposed rats and the blood of Cd exposed workers. Cadmium 230-232 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 160-166 30090505-6 2017 A significant positive correlation was observed between blood MALAT1 expression and urinary/blood Cd concentrations, and there were significant correlations of MALAT1 expression with the expressions of target genes in the lung of Cd-exposed rats and the blood of Cd exposed workers. Cadmium 230-232 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 62-68 30090505-6 2017 A significant positive correlation was observed between blood MALAT1 expression and urinary/blood Cd concentrations, and there were significant correlations of MALAT1 expression with the expressions of target genes in the lung of Cd-exposed rats and the blood of Cd exposed workers. Cadmium 230-232 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 160-166 30090505-7 2017 This study suggests that the expression of MALAT1 is upregulated and regulates the cell cycle progression, proliferation, apoptosis, migration and invasion in Cd toxicity. Cadmium 159-161 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 43-49 30090505-8 2017 MALAT1 may serve as a novel valuable biomarker of cadmium exposure and cadmium toxicity. Cadmium 50-57 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 0-6 30090505-8 2017 MALAT1 may serve as a novel valuable biomarker of cadmium exposure and cadmium toxicity. Cadmium 71-78 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 0-6 25679300-4 2015 The KMC simulations carried out using singly deuterated VCN (CH2=CD-CN) at 148 kcal mol(-1) show the importance of hydrogen-deuterium exchange reactions: both DCN and HCN will be produced in any of the 1,1 and 1,2 elimination pathways. Cadmium 65-67 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 167-170