PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23434795-5	2013	In lean mice, TNFR2 deficiency ablated O(3)-induced AHR without affecting pulmonary inflammation; whereas in obese mice, TNFR2 deficiency augmented O(3)-induced AHR but reduced inflammatory cell recruitment.	Ozone	148-152	aryl-hydrocarbon receptor	Mus musculus	161-164	
23434795-1	2013	BACKGROUND: Acute ozone (O(3)) exposure results in greater inflammation and airway hyperresponsiveness (AHR) in obese versus lean mice.	Ozone	25-29	aryl-hydrocarbon receptor	Mus musculus	104-107	
23434795-5	2013	In lean mice, TNFR2 deficiency ablated O(3)-induced AHR without affecting pulmonary inflammation; whereas in obese mice, TNFR2 deficiency augmented O(3)-induced AHR but reduced inflammatory cell recruitment.	Ozone	39-43	aryl-hydrocarbon receptor	Mus musculus	52-55	
15516495-7	2005	Acute O(3) exposure caused AHR in both groups of mice, but there was no genotype-related difference in the magnitude of O(3)-induced AHR.	Ozone	6-10	aryl-hydrocarbon receptor	Mus musculus	27-30	
12794034-7	2003	Exposure to O3 (2 parts/million for 3 h) caused AHR and airway inflammation in both groups of mice, but responses to O3 were enhanced in ob/ob compared with wild-type mice.	Ozone	12-14	aryl-hydrocarbon receptor	Mus musculus	48-51