PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30236892-4	2018	We utilized U251 and U87 human tumor cells to understand VM in an orthotopic GBM model and AAT-mediated enhancement in VM was modeled using vatalanib (anti-VEGFR2) and avastin (anti-VEGF).	vatalanib	140-149	serpin family A member 1	Homo sapiens	91-94	
27990624-4	2017	Here, we identified that GBM following AAT (Vatalanib) acquired an alternate mechanism to support tumor growth, called vascular mimicry (VM).	vatalanib	44-53	serpin family A member 1	Homo sapiens	39-42