PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28424220-0	2017	GABABR-Induced EGFR Transactivation Promotes Migration of Human Prostate Cancer Cells.	gababr	0-6	epidermal growth factor receptor	Homo sapiens	15-19	
28424220-5	2017	RTK array analysis revealed that the GABABR-specific agonist baclofen selectively induced the transactivation of EGFR in PC-3 cells.	gababr	37-43	epidermal growth factor receptor	Homo sapiens	113-117	
28424220-7	2017	Positive allosteric modulators (PAMs) of GABABR, such as CGP7930, rac-BHFF, and GS39783, can function as PAM agonists to induce EGFR transactivation and subsequent ERK1/2 activation.	gababr	41-47	epidermal growth factor receptor	Homo sapiens	128-132	
28424220-9	2017	In summary, our observations demonstrated that GABABR transactivated EGFR in a ligand-dependent mechanism to promote prostate cancer cell migration and invasion, thus providing new insights into developing a novel strategy for prostate cancer treatment by targeting neurotransmitter signaling.	gababr	47-53	epidermal growth factor receptor	Homo sapiens	69-73