PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33432187-5	2021	Using a novel technique, we show that synapse formation underlying the behavioral effects of Ro-25-6981 requires GABABR-mediated mTORC1 activity in WT animals.	gababr	113-119	CREB regulated transcription coactivator 1	Mus musculus	129-135	
23752093-0	2013	mTORC1-dependent protein synthesis underlying rapid antidepressant effect requires GABABR signaling.	gababr	83-89	CREB regulated transcription coactivator 1	Mus musculus	0-6	
23752093-5	2013	In vivo, blocking GABABR prevents the fast-acting, anti-depressant effect of the NR2B antagonist, Ro-25-6891, decreases active mTORC1 kinase, and reduces expression of BDNF and the AMPA receptor subunit GluA1.	gababr	18-24	CREB regulated transcription coactivator 1	Mus musculus	127-133	
29133952-8	2018	Recent studies notably demonstrate that a shift in gamma-amino-butyric acid receptor B (GABABR) function, from inhibitory to excitatory, is required for mTORC1-dependent translation with NMDAR antagonists.	gababr	88-94	CREB regulated transcription coactivator 1	Mus musculus	153-159	
29133952-9	2018	Finally, we discuss how GABABR activation of mTORC1 helps resolve key discrepancies between rapid-acting antidepressants and local homeostatic mechanisms.	gababr	24-30	CREB regulated transcription coactivator 1	Mus musculus	45-51