PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21556141-8	2011	Furthermore, blocking K(+) efflux protected Tg2576 neurons from Abeta-induced neurotoxicity.	tg2576	44-50	amyloid beta precursor protein	Homo sapiens	64-69	
16828889-3	2006	Aged F-344 rats and young mice over-expressing the Swedish mutation in the human amyloid precursor protein (APPsw; Tg2576+) had elevated levels of soluble Abeta, and were more vulnerable to scopolamine challenge in the Morris water maze (MWM), relative to young rats and Tg2576- mice; but, among individual animals, higher levels of soluble Abeta were not correlated with vulnerability to scopolamine.	tg2576	115-121	amyloid beta precursor protein	Homo sapiens	81-106	
16828889-3	2006	Aged F-344 rats and young mice over-expressing the Swedish mutation in the human amyloid precursor protein (APPsw; Tg2576+) had elevated levels of soluble Abeta, and were more vulnerable to scopolamine challenge in the Morris water maze (MWM), relative to young rats and Tg2576- mice; but, among individual animals, higher levels of soluble Abeta were not correlated with vulnerability to scopolamine.	tg2576	115-121	amyloid beta precursor protein	Homo sapiens	155-160	
15653168-6	2005	Peripheral administration of these vaccines augmented humoral responses to Abeta and reduced CNS Abeta deposition in Tg2576 AD mice.	tg2576	117-123	amyloid beta precursor protein	Homo sapiens	97-102	
11325345-1	2001	A transgenic mouse expressing the human beta-amyloid precursor protein with the "Swedish" mutation, Tg2576, was used to investigate the mechanism of beta-amyloid (Abeta) deposition.	tg2576	100-106	amyloid beta precursor protein	Homo sapiens	40-70	
11377835-1	2001	A transgenic mouse expressing the human beta-amyloid precursor protein with the "Swedish" mutation, Tg2576, was used to investigate the mechanism of amyloid-beta peptide (Abeta) deposition.	tg2576	100-106	amyloid beta precursor protein	Homo sapiens	40-70	
29997495-8	2018	We extended this line of research to in vivo studies using transgenic human amyloid precursor protein (hAPP)-overexpressing mice (Tg2576) that were treated with PYR41, a cell-permeable, irreversible inhibitor of the ubiquitin-activating enzyme E1.	tg2576	130-136	amyloid beta precursor protein	Homo sapiens	103-107