PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29508777-1	1998	It has been identified that the expansion of normally polymorphic CTG repeats in myotonin protein kinase (DM-PK) gene in myotonic dystrophy (DM) and CAG repeats in huntingtin gene in Huntington"s disease (HD) acts as the causative mutation.	ctg	66-69	huntingtin	Homo sapiens	164-174	
23157165-10	2012	This work highlights the innate ability of CAG/CTG repeats to incorporate and to position in nucleosomes and how that behavior is modulated by the htt flanking sequence.	ctg	47-50	huntingtin	Homo sapiens	147-150	
34520257-3	2021	HD is caused by a CAG CTG trinucleotide-repeat expansion in exon 1 of the huntingtin (HTT) gene leading to the formation of mutant HTT (mtHTT) protein aggregates.	ctg	22-25	huntingtin	Homo sapiens	74-84	
34520257-3	2021	HD is caused by a CAG CTG trinucleotide-repeat expansion in exon 1 of the huntingtin (HTT) gene leading to the formation of mutant HTT (mtHTT) protein aggregates.	ctg	22-25	huntingtin	Homo sapiens	86-89	
34520257-3	2021	HD is caused by a CAG CTG trinucleotide-repeat expansion in exon 1 of the huntingtin (HTT) gene leading to the formation of mutant HTT (mtHTT) protein aggregates.	ctg	22-25	huntingtin	Homo sapiens	131-134