PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17097285-9	2007	We found that celecoxib and PSC833, but not indomethacin or NS398, almost completely inhibited basal- and dox induced NF-kappaB gene-reporter activity and p65 subunit nuclear translocation.	valspodar	28-34	nuclear factor kappa B subunit 1	Homo sapiens	118-127	
17097285-10	2007	Furthermore, the NF-kappaB inhibitor PDTC mimicked the actions of celecoxib and PSC833 on cell growth and on intracellular accumulation of dox, suggesting that NF-kappaB is functionally involved in the actions of these compounds.	valspodar	80-86	nuclear factor kappa B subunit 1	Homo sapiens	17-26	
17097285-10	2007	Furthermore, the NF-kappaB inhibitor PDTC mimicked the actions of celecoxib and PSC833 on cell growth and on intracellular accumulation of dox, suggesting that NF-kappaB is functionally involved in the actions of these compounds.	valspodar	80-86	nuclear factor kappa B subunit 1	Homo sapiens	160-169	
17097285-11	2007	In conclusion, we show that structurally different compounds, among which are celecoxib and PSC833, increase the intracellular accumulation of dox and enhance dox induced cytotoxicity in MDA-MB231 breast cancer cells most likely via the modulation of NF-kappaB activity.	valspodar	92-98	nuclear factor kappa B subunit 1	Homo sapiens	251-260