PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30612190-6	2019	In support of the essential cytokine contribution in the AR-agonist induced SAA production is the fact that the anti-inflammatory drug, sodium salicylate, prevented the AR-stimulated hepatic SAA1/2 synthesis by reducing IL-1beta levels, whereas IL-1beta inhibition with Anakinra mimics this sodium salicylate preventive effect, thus indicating a crucial role for IL-1beta.	Sodium Salicylate	136-153	interleukin 1 beta	Mus musculus	220-228	
30612190-6	2019	In support of the essential cytokine contribution in the AR-agonist induced SAA production is the fact that the anti-inflammatory drug, sodium salicylate, prevented the AR-stimulated hepatic SAA1/2 synthesis by reducing IL-1beta levels, whereas IL-1beta inhibition with Anakinra mimics this sodium salicylate preventive effect, thus indicating a crucial role for IL-1beta.	Sodium Salicylate	136-153	interleukin 1 beta	Mus musculus	245-253	
30612190-6	2019	In support of the essential cytokine contribution in the AR-agonist induced SAA production is the fact that the anti-inflammatory drug, sodium salicylate, prevented the AR-stimulated hepatic SAA1/2 synthesis by reducing IL-1beta levels, whereas IL-1beta inhibition with Anakinra mimics this sodium salicylate preventive effect, thus indicating a crucial role for IL-1beta.	Sodium Salicylate	136-153	interleukin 1 beta	Mus musculus	245-253	
8590986-12	1995	was significantly blocked by pretreatment with sodium salicylate (a cyclo-oxygenase inhibitor, 10 ng or 30 ng/mouse) administered intracisternally 15 min before IL-1 beta, while i.c.	Sodium Salicylate	47-64	interleukin 1 beta	Mus musculus	161-170