PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 10574634-4 1999 In contrast, sodium salicylate (NaSal), a nonsteroidal anti-inflammatory drug (NSAID), could activate p38 MAPK but not ERK within one hour. Sodium Salicylate 13-30 mitogen-activated protein kinase 14 Homo sapiens 102-105 16393981-6 2006 For example, sodium salicylate blocked GM-CSF-stimulated Erk and Akt activation, but resulted in rapid and sustained activation of p38-MAPK, an event mimicked by okadaic acid that also accelerates Mcl-1 turnover and neutrophil apoptosis. Sodium Salicylate 13-30 mitogen-activated protein kinase 14 Homo sapiens 131-134 10574634-4 1999 In contrast, sodium salicylate (NaSal), a nonsteroidal anti-inflammatory drug (NSAID), could activate p38 MAPK but not ERK within one hour. Sodium Salicylate 32-37 mitogen-activated protein kinase 14 Homo sapiens 102-105 10082138-2 1999 On the other hand, we recently showed that in normal human diploid fibroblasts sodium salicylate (NaSal) elicits activation of p38 MAPK but not activation of c-Jun N-terminal kinase (JNK). Sodium Salicylate 79-96 mitogen-activated protein kinase 14 Homo sapiens 127-130 9418855-0 1998 Activation of p38 mitogen-activated protein kinase by sodium salicylate leads to inhibition of tumor necrosis factor-induced IkappaB alpha phosphorylation and degradation. Sodium Salicylate 54-71 mitogen-activated protein kinase 14 Homo sapiens 14-17 9096313-0 1997 Sodium salicylate induces apoptosis via p38 mitogen-activated protein kinase but inhibits tumor necrosis factor-induced c-Jun N-terminal kinase/stress-activated protein kinase activation. Sodium Salicylate 0-17 mitogen-activated protein kinase 14 Homo sapiens 40-43 9096313-5 1997 NaSal-induced apoptosis was blocked by the selective p38 MAPK inhibitor SB-203580, indicating that p38 MAPK serves as a mediator of NaSal-induced apoptosis in human fibroblasts. Sodium Salicylate 0-5 mitogen-activated protein kinase 14 Homo sapiens 53-56 9096313-5 1997 NaSal-induced apoptosis was blocked by the selective p38 MAPK inhibitor SB-203580, indicating that p38 MAPK serves as a mediator of NaSal-induced apoptosis in human fibroblasts. Sodium Salicylate 0-5 mitogen-activated protein kinase 14 Homo sapiens 99-102 24692721-0 2014 Potent reactive oxygen species-JNK-p38 activation by sodium salicylate potentiates death of primary effusion lymphoma cells. Sodium Salicylate 53-70 mitogen-activated protein kinase 14 Homo sapiens 35-38