PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16393981-4	2006	Sodium salicylate resulted in rapid activation of caspase-3, -8, -9, and -10, and salicylate-accelerated Mcl-1 turnover was partly blocked by caspase inhibitors.	Sodium Salicylate	0-17	caspase 3	Homo sapiens	50-76	
12851702-3	2003	Here we show that in HCT116 colon carcinoma cells, 10 mM sodium salicylate induces caspase-3 activation and degradation of its substrates, poly(ADP-ribose) polymerase (PARP), beta-catenin, and retinoblastoma (Rb).	Sodium Salicylate	57-74	caspase 3	Homo sapiens	83-92	
10090950-3	1999	TF-1 cells undergo rapid apoptosis on treatment with Na-Sal, as indicated by increased annexin V binding capacity, cpp-32 (caspase-3) activation, and cleavage of poly (ADP-ribose) polymerase (PARP) and gelsolin.	Sodium Salicylate	53-59	caspase 3	Homo sapiens	115-121	
10090950-3	1999	TF-1 cells undergo rapid apoptosis on treatment with Na-Sal, as indicated by increased annexin V binding capacity, cpp-32 (caspase-3) activation, and cleavage of poly (ADP-ribose) polymerase (PARP) and gelsolin.	Sodium Salicylate	53-59	caspase 3	Homo sapiens	123-132