PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16623712-5	2006	In particular, the fluorogenic substrate Mca-Ala-Ser-Asp-Lys-DpaOH was shown to be a strict N-domain-selective substrate of mouse ACE, whereas with rat ACE it displayed marked C-domain selectivity.	Aspartic Acid	53-57	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	130-133	
14757757-6	2004	Analysis of homozygous mutant mice (termed ACE 7/7) showed normal plasma levels of angiotensin II but an elevation of plasma and urine N-acetyl-Ser-Asp-Lys-Pro, a peptide suggested to inhibit bone marrow maturation.	Aspartic Acid	148-151	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	43-46	
6101060-1	1983	The brain and lung angiotensin converting enzyme (ACE) activities of the mice injected with 10 mg/kg morphine and/or naloxone, 200 mg/kg D- and/or L-aspartic acid were spectrophotometrically determined.	Aspartic Acid	149-162	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	50-53	
6101060-2	1983	Morphine, naloxone, D- and L-aspartic acid alone inhibited both brain and lung ACE activities whereas the combinations of morphine with naloxone, D-aspartic acid with L-aspartic acid and morphine with naloxone + L-aspartic acid showed no inhibitory effect on the brain ACE.	Aspartic Acid	27-42	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	79-82	
6101060-3	1983	While naloxone or L-aspartic acid partly antagonized the suppression of morphine on the lung ACE their combination completely prevented morphine from inhibiting the lung ACE.	Aspartic Acid	18-33	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	93-96	
6101060-3	1983	While naloxone or L-aspartic acid partly antagonized the suppression of morphine on the lung ACE their combination completely prevented morphine from inhibiting the lung ACE.	Aspartic Acid	18-33	angiotensin I converting enzyme (peptidyl-dipeptidase A) 1	Mus musculus	170-173