PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28562331-7	2017	On GLI1, substitution of AMPK phosphorylation sites to aspartic acid (GLI13E) results in stronger binding affinity of GLI1 with beta-TrCP, accompanied by enhanced GLI1 ubiquitination and later degradation.	Aspartic Acid	55-68	protein kinase AMP-activated non-catalytic subunit beta 1	Homo sapiens	25-29	
25792737-5	2015	Substitution of Thr-148 for a phospho-mimicking aspartate (T148D) prevents AMPK from binding to carbohydrate.	Aspartic Acid	48-57	protein kinase AMP-activated non-catalytic subunit beta 1	Homo sapiens	75-79