PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25395009-7	2015	In this study, we investigated whether the alkylating agent 2-chloroethyl-ethylsulfide (CEES, a model substance for SM-promoted effects) and SM are able to activate TRPA1 channels.	2-chloroethyl ethyl sulfide	60-86	transient receptor potential cation channel subfamily A member 1	Homo sapiens	165-170	
27738742-5	2017	Previous studies already revealed an impact of 2-chloroethyl-ethyl sulfide on the ion channel TRPA1.	2-chloroethyl ethyl sulfide	47-74	transient receptor potential cation channel subfamily A member 1	Homo sapiens	94-99	
25395009-7	2015	In this study, we investigated whether the alkylating agent 2-chloroethyl-ethylsulfide (CEES, a model substance for SM-promoted effects) and SM are able to activate TRPA1 channels.	2-chloroethyl ethyl sulfide	88-92	transient receptor potential cation channel subfamily A member 1	Homo sapiens	165-170	
25395009-8	2015	CEES induced a marked increase in the intracellular calcium concentration ([Ca(2+)]i) in TRPA1-expressing but not in TRPA1-negative cells.	2-chloroethyl ethyl sulfide	0-4	transient receptor potential cation channel subfamily A member 1	Homo sapiens	89-94	
25395009-10	2015	HEK293 cells permanently expressing TRPA1 were more sensitive toward cytotoxic effects of CEES compared with wild-type cells.	2-chloroethyl ethyl sulfide	90-94	transient receptor potential cation channel subfamily A member 1	Homo sapiens	36-41	
25395009-13	2015	Human A549 lung epithelial cells, which express TRPA1 endogenously, reacted with a transient calcium influx in response to CEES exposure.	2-chloroethyl ethyl sulfide	123-127	transient receptor potential cation channel subfamily A member 1	Homo sapiens	48-53