PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17785780-3	2007	Treatment of SpDC with laminarin or glucan phosphate, two molecules known to block Dectin-1-dependent activity, led to a reduction in M. tuberculosis-induced IL-12p40 as well as IL-12p70 production.	laminaran	23-32	C-type lectin domain family 7, member a	Mus musculus	83-91	
33705858-11	2021	To determine whether the antidepressant effect was mediated by Dectin-1, we found that GLP treatment enhanced Dectin-1 expression in the hippocampus in CSDS mice, and the Dectin-1-specific inhibitor laminarin almost completely blocked the antidepressant effect of GLP.	laminaran	199-208	C-type lectin domain family 7, member a	Mus musculus	63-71	
33705858-11	2021	To determine whether the antidepressant effect was mediated by Dectin-1, we found that GLP treatment enhanced Dectin-1 expression in the hippocampus in CSDS mice, and the Dectin-1-specific inhibitor laminarin almost completely blocked the antidepressant effect of GLP.	laminaran	199-208	C-type lectin domain family 7, member a	Mus musculus	110-118	
33705858-11	2021	To determine whether the antidepressant effect was mediated by Dectin-1, we found that GLP treatment enhanced Dectin-1 expression in the hippocampus in CSDS mice, and the Dectin-1-specific inhibitor laminarin almost completely blocked the antidepressant effect of GLP.	laminaran	199-208	C-type lectin domain family 7, member a	Mus musculus	110-118	
31926564-9	2020	The Dectin-1 antagonist LAM or Syk inhibitor PIC decreased the number of Iba1-positive cells and TNF-alpha and iNOS expression, decreased the brain infarct volume, and improved neurological functions on day 3 after ischemic stroke.	laminaran	24-27	C-type lectin domain family 7, member a	Mus musculus	4-12	
21424514-6	2011	Observations based on injection of curdlan, laminarin or mannan supported the dominant role of the Dectin-1 pathway in the joint.	laminaran	44-53	C-type lectin domain family 7, member a	Mus musculus	99-107	
28326085-5	2017	With that intent, we observed that the absence of Dectin-1, observed in genetically depleted (Dectin-1-/-) mice or competitively inhibited by an inert agonist [laminarin (LAM)], rescued 50% of the mice infected with lethal doses of N. caninum.	laminaran	160-169	C-type lectin domain family 7, member a	Mus musculus	50-58	
28326085-5	2017	With that intent, we observed that the absence of Dectin-1, observed in genetically depleted (Dectin-1-/-) mice or competitively inhibited by an inert agonist [laminarin (LAM)], rescued 50% of the mice infected with lethal doses of N. caninum.	laminaran	171-174	C-type lectin domain family 7, member a	Mus musculus	50-58	
28326085-9	2017	We also found that Dectin-1+ cells produced less reactive oxygen species (ROS) at the initial site of the infection, while mice deficient in NADPH oxidase isoform 2 (NOX2-/-) were not able to control parasite replication and produce IL-12p40, even upon LAM treatment.	laminaran	253-256	C-type lectin domain family 7, member a	Mus musculus	19-27	
19327548-8	2009	Addition of laminarin, a well-established dectin-1 inhibitor, significantly inhibited surface expression of dectin-1 (p < 0.05), which further confirmed that dectin-1 surface expression was up-regulated by non-opsonized zymosan activation.	laminaran	12-21	C-type lectin domain family 7, member a	Mus musculus	42-50	
19327548-8	2009	Addition of laminarin, a well-established dectin-1 inhibitor, significantly inhibited surface expression of dectin-1 (p < 0.05), which further confirmed that dectin-1 surface expression was up-regulated by non-opsonized zymosan activation.	laminaran	12-21	C-type lectin domain family 7, member a	Mus musculus	108-116	
19327548-8	2009	Addition of laminarin, a well-established dectin-1 inhibitor, significantly inhibited surface expression of dectin-1 (p < 0.05), which further confirmed that dectin-1 surface expression was up-regulated by non-opsonized zymosan activation.	laminaran	12-21	C-type lectin domain family 7, member a	Mus musculus	108-116