PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33995824-0	2021	Vinegar/Tetramethylpyrazine Induces Nutritional Preconditioning Protecting the Myocardium Mediated by VDAC1.	Acetic Acid	0-7	voltage-dependent anion channel 1	Mus musculus	102-107	
33995824-9	2021	However, the protective effects of vinegar/TMP pretreatment were accompanied by the downregulation of VDAC1 expression in the myocardium and reversed by pAD/VDAC1, an adenovirus that upregulates VDAC1 expression.	Acetic Acid	35-42	voltage-dependent anion channel 1	Mus musculus	102-107	
33995824-9	2021	However, the protective effects of vinegar/TMP pretreatment were accompanied by the downregulation of VDAC1 expression in the myocardium and reversed by pAD/VDAC1, an adenovirus that upregulates VDAC1 expression.	Acetic Acid	35-42	voltage-dependent anion channel 1	Mus musculus	157-162	
33995824-9	2021	However, the protective effects of vinegar/TMP pretreatment were accompanied by the downregulation of VDAC1 expression in the myocardium and reversed by pAD/VDAC1, an adenovirus that upregulates VDAC1 expression.	Acetic Acid	35-42	voltage-dependent anion channel 1	Mus musculus	157-162	
33995824-10	2021	In conclusion, this study is the first to demonstrate that vinegar/TMP pretreatment could induce myocardial protection of NPC due to downregulating VDAC1 expression, inhibiting oxidative stress, and preventing mitochondrial dysfunction; that is, VDAC1 is their target, and the mitochondria are their target organelles.	Acetic Acid	59-66	voltage-dependent anion channel 1	Mus musculus	148-153	
33995824-10	2021	In conclusion, this study is the first to demonstrate that vinegar/TMP pretreatment could induce myocardial protection of NPC due to downregulating VDAC1 expression, inhibiting oxidative stress, and preventing mitochondrial dysfunction; that is, VDAC1 is their target, and the mitochondria are their target organelles.	Acetic Acid	59-66	voltage-dependent anion channel 1	Mus musculus	246-251