PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30132829-7	2018	Preincubation of rat ASMCs with pioglitazone, a PPAR-gamma activator, blocked TGF-beta1-induced activation of Smad2/3 and its downstream targets changes of miR-21, PTEN, Akt, FOXO1, and cyclin D1, resulting in the inhibition of rat ASMC proliferation.	asmcs	21-26	transforming growth factor, beta 1	Rattus norvegicus	78-87	
11348874-9	2001	In contrast, the growth response of decorin-overexpressing ASMCs to TGF-beta1, as well as the expression of TGF-beta1-responsive genes, such as plasminogen activator inhibitor-1 and versican (an extracellular matrix proteoglycan), was diminished.	asmcs	59-64	transforming growth factor, beta 1	Rattus norvegicus	68-77	
11348874-10	2001	These results indicate that decorin selectively inhibits the responsiveness of rat ASMCs to TGF-beta1 and suggests that the induction of constitutive decorin overexpression by ASMCs in vivo may have therapeutic value in the inhibition of TGF-beta1-mediated effects on the development of atherosclerotic lesions.	asmcs	83-88	transforming growth factor, beta 1	Rattus norvegicus	92-101	
29949788-13	2018	CONCLUSION: The results of our study suggest that miR-142 inhibits proliferation and promotes apoptosis in ASMCs during airway remodeling in asthmatic rats by inhibiting TGF-beta expression via a mechanism involving the EGFR signaling pathway.	asmcs	107-112	transforming growth factor, beta 1	Rattus norvegicus	170-178	
25109503-8	2014	RESULTS: Compared with normal control, TGF-beta1 secretion was significantly increased in asthmatic ASMCs; meanwhile, TGF-beta1 promoted ASMCs proliferation (P < 0.05).	asmcs	100-105	transforming growth factor, beta 1	Rattus norvegicus	39-48	
22270542-2	2012	Urotensin II (UII) and transforming growth factor (TGF)-beta are potent mitogens for ASMCs proliferation.	asmcs	85-90	transforming growth factor, beta 1	Rattus norvegicus	51-60	
22270542-3	2012	The study was aimed to determine whether UII-upregulated TGF-beta-mediated ASMCs proliferation and extracellular signal-regulated kinase (ERK) was required for such an effect.	asmcs	75-80	transforming growth factor, beta 1	Rattus norvegicus	57-65	
22270542-7	2012	In vitro experiments were conducted to determine the direct effect of UII on TGF-beta1 expression by ASMCs.	asmcs	101-106	transforming growth factor, beta 1	Rattus norvegicus	77-86	
22270542-10	2012	In vitro study on ASMCs further revealed that UII may render its effect on ASMCs cells through the upregulation of TGF-beta1.	asmcs	18-23	transforming growth factor, beta 1	Rattus norvegicus	115-124	
22270542-12	2012	The current study provides new evidence that UII is involved in the TGF-beta mediated mitogenic effect on ASMCs.	asmcs	106-111	transforming growth factor, beta 1	Rattus norvegicus	68-76