PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26916957-6	2016	Platelet-derived growth factor (PDGF)-BB was used to induce the proliferation of isolated ASMCs, and the expression of miR-135a and TRPC1 was detected in PDGF-BB-treated ASMCs.	asmcs	170-175	transient receptor potential cation channel, subfamily C, member 1	Rattus norvegicus	132-137	
26916957-11	2016	In addition, Schisandrin B reversed the expression of miR-135a and TRPC1 in PDGF-BB-induced ASMCs.	asmcs	92-97	transient receptor potential cation channel, subfamily C, member 1	Rattus norvegicus	67-72	
26916957-12	2016	Si-TRPC1 abrogated the increasing proliferation of ASMCs induced by miR-135a inhibitor.	asmcs	51-56	transient receptor potential cation channel, subfamily C, member 1	Rattus norvegicus	3-8	
26916957-14	2016	These results demonstrate that Schisandrin B inhibits the proliferation of ASMCs via miR-135a suppressing the expression of TRPC1.	asmcs	75-80	transient receptor potential cation channel, subfamily C, member 1	Rattus norvegicus	124-129	
19897708-6	2010	This reflected an augmented expression of 2.5-fold in OH ASMCs of C-type transient receptor potential TRPC1, an essential component of store-operated channels (SOCs); two other components of some SOCs were not expressed (TRPC4) or were not upregulated (TRPC5).	asmcs	57-62	transient receptor potential cation channel, subfamily C, member 1	Rattus norvegicus	102-107	
27648131-10	2016	In addition,TRPC1 overexpression reversed the function of si-BCYRN1 indecreasing the viability/proliferation and migration of ASMCs treated with PDGF-BB.	asmcs	126-131	transient receptor potential cation channel, subfamily C, member 1	Rattus norvegicus	12-17