PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35237235-8	2022	By using age- and pentosidine-matched normoglycemic individuals as references, the PBMC-isolated from prediabetic patients demonstrated lower expression of both AGER and BAX/BCL2.	pentosidine	18-29	advanced glycosylation end-product specific receptor	Homo sapiens	161-165	
18448811-5	2008	Pentosidine incubation of HOb caused a significant decrease in ALP, Col Ialpha1, and RAGE mRNA levels, but only the RAGE gene expression decreased with no dose dependency.	pentosidine	0-11	advanced glycosylation end-product specific receptor	Homo sapiens	85-89	
18325460-10	2008	In the multivariate Cox proportional hazard analysis, soluble RAGE and serum pentosidine were independent risk factors for cardiac events (soluble RAGE: HR 1.90, 95% CI 1.16-3.09, P = .010; pentosidine: HR 1.59, 95% CI 1.11-2.29, P = .012).	pentosidine	190-201	advanced glycosylation end-product specific receptor	Homo sapiens	62-66	
15213278-5	2004	RAGE expression on peripheral blood monocytes increased with worsening CKD (r2 = 0.73) and was strongly correlated with plasma levels of pentosidine, a marker for AGE (r = 0.71).	pentosidine	137-148	advanced glycosylation end-product specific receptor	Homo sapiens	0-4	
35237235-10	2022	The multivariate analysis demonstrated that serum pentosidine is an independent risk factor for AGER expression.	pentosidine	50-61	advanced glycosylation end-product specific receptor	Homo sapiens	96-100	
35237235-11	2022	With logistic regression analysis, the area under the ROC curve (AUC) for serum pentosidine at the cut-off level of 2.1 ng/ml and FPG at 100 mg/dl, which is a cut-off point for prediabetes, was significantly higher for predicting AGER expression than that of serum pentosidine alone (0.803 vs 0.688, p = 0.048), indicating that serum pentosidine was a good predictor of AGER expression in prediabetic individuals.	pentosidine	80-91	advanced glycosylation end-product specific receptor	Homo sapiens	230-234	
35237235-11	2022	With logistic regression analysis, the area under the ROC curve (AUC) for serum pentosidine at the cut-off level of 2.1 ng/ml and FPG at 100 mg/dl, which is a cut-off point for prediabetes, was significantly higher for predicting AGER expression than that of serum pentosidine alone (0.803 vs 0.688, p = 0.048), indicating that serum pentosidine was a good predictor of AGER expression in prediabetic individuals.	pentosidine	80-91	advanced glycosylation end-product specific receptor	Homo sapiens	370-374	
35237235-14	2022	Serum pentosidine was an independent risk factor for cellular RAGE expression and is conceivably a good predictor for AGER suppression in prediabetic individuals.	pentosidine	6-17	advanced glycosylation end-product specific receptor	Homo sapiens	62-66	
35237235-14	2022	Serum pentosidine was an independent risk factor for cellular RAGE expression and is conceivably a good predictor for AGER suppression in prediabetic individuals.	pentosidine	6-17	advanced glycosylation end-product specific receptor	Homo sapiens	118-122	
20638753-6	2012	We showed that pentosidine and GLAP were able to upregulate BACE1 expression through their binding with RAGE and the consequent activation of NF-kappaB.	pentosidine	15-26	advanced glycosylation end-product specific receptor	Homo sapiens	104-108