PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22497476-6	2012	Olmesartan treatment significantly reduced fibrosis, hypertrophy and their marker molecules (OPN, CTGF, ANP and GATA-4, respectively), as well as matrix metalloproteinases compared with those of vehicle-treated DCM rats.	olmesartan	0-10	cellular communication network factor 2	Rattus norvegicus	98-102	
20944640-0	2010	Connective tissue growth factor induction in a pressure-overloaded heart ameliorated by the angiotensin II type 1 receptor blocker olmesartan.	olmesartan	131-141	cellular communication network factor 2	Rattus norvegicus	0-31	
20944640-11	2010	Finally, olmesartan ameliorated the cardiac dysfunction in CTGF- and TGF-beta1-infused rats.	olmesartan	9-19	cellular communication network factor 2	Rattus norvegicus	59-63	
20944640-12	2010	Olmesartan attenuated CTGF induction, reduced perivascular fibrosis and ameliorated cardiac dysfunction in a PO heart.	olmesartan	0-10	cellular communication network factor 2	Rattus norvegicus	22-26	
16755152-10	2006	Olmesartan at 10 mg/kg/day tended to decrease renal cortical and medullary Ang II levels, TGF-beta and CTGF expression, and collagen contents; however, these changes were not significant.	olmesartan	0-10	cellular communication network factor 2	Rattus norvegicus	103-107