PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33660877-8	2021	In CHD patients, lncRNA MALAT1 was positively correlated with Gensini score, total cholesterol, low-density lipoprotein cholesterol, C-reactive protein, tumor necrosis factor alpha, interleukin (IL)-1beta, IL-6, IL-17, and accumulating MACE occurrence; reversely, miR-125b presented a opposite trend.	Cholesterol	83-94	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	24-30	
32346662-10	2020	Additionally, the principal component analyzes showed that serum MALAT1 expression was associated with total cholesterol and HDL-c levels, and the exosomes H19 expression was associated with waist circumference.	Cholesterol	109-120	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	65-71	
33660877-8	2021	In CHD patients, lncRNA MALAT1 was positively correlated with Gensini score, total cholesterol, low-density lipoprotein cholesterol, C-reactive protein, tumor necrosis factor alpha, interleukin (IL)-1beta, IL-6, IL-17, and accumulating MACE occurrence; reversely, miR-125b presented a opposite trend.	Cholesterol	120-131	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	24-30	
33565328-3	2021	Results: Lnc-MALAT1 negatively correlated with miR-125b, miR-146a and miR-203 Furthermore, lnc-MALAT1, miR-125b, miR-146a and miR-203 correlated with diabetes mellitus, hyperuricemia, lesion properties, cholesterol, inflammation and cardiac function indexes.	Cholesterol	203-214	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	13-19	
33565328-3	2021	Results: Lnc-MALAT1 negatively correlated with miR-125b, miR-146a and miR-203 Furthermore, lnc-MALAT1, miR-125b, miR-146a and miR-203 correlated with diabetes mellitus, hyperuricemia, lesion properties, cholesterol, inflammation and cardiac function indexes.	Cholesterol	203-214	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	95-101	
26404789-0	2015	Isoform dependent regulation of human HCN channels by cholesterol.	Cholesterol	54-65	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	38-41	
32319624-0	2020	Long non-coding RNA MALAT1 regulates cholesterol accumulation in ox-LDL-induced macrophages via the microRNA-17-5p/ABCA1 axis.	Cholesterol	37-48	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	20-26	
32319624-3	2020	However, whether MALAT1 can affect cholesterol accumulation in macrophages during AS progression, and the potential molecular mechanism involved in this progression have not been elucidated.	Cholesterol	35-46	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	17-23	
32319624-8	2020	Knockdown of MALAT1 increased ox-LDL uptake, lipid accumulation and the total cholesterol (T-CHO) level in ox-LDL-induced macrophages.	Cholesterol	78-89	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	13-19	
32319624-11	2020	Subsequently, the present study investigated whether MALAT1 could target miR-17-5p to regulate the expression level of ABCA1, which is involved in cholesterol efflux from macrophages.	Cholesterol	147-158	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	53-59	
32319624-13	2020	In summary, knockdown of MALAT1 may promote cholesterol accumulation by regulating the miR-17-5p/ABCA1 axis in ox-LDL-induced THP-1 macrophages.	Cholesterol	44-55	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	25-31