PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 11979500-13 2002 In contrast to insulin single therapy, vitamin A alone significantly prevented an increase in catalase activity of diabetic heart, and a combination of these agents did not supply any further benefit. Vitamin A 39-48 catalase Rattus norvegicus 94-102 14734217-12 2004 Post stress vitamin E treatment was found most effective than vitamins A and C in enhancing the levels of glutathione and activities of SOD, GST and catalase and decreasing lipid peroxidation. Vitamin A 62-72 catalase Rattus norvegicus 149-157 23485553-0 2014 Daily rhythms of catalase and glutathione peroxidase expression and activity are endogenously driven in the hippocampus and are modified by a vitamin A-free diet. Vitamin A 142-151 catalase Rattus norvegicus 17-25 11328675-3 2001 We report here that retinol caused cellular oxidative stress and modulated superoxide dismutase, catalase and glutathione peroxidase activities. Vitamin A 20-27 catalase Rattus norvegicus 97-105 11328675-5 2001 In response to retinol treatment superoxide dismutase, catalase and glutathione peroxidase activities increased. Vitamin A 15-22 catalase Rattus norvegicus 55-63 969379-1 1976 A long-term administration of retinol in a dose exceeding 15-fold the diurnal requirement to rats weighing 170-200 g provoked a diminution of the erythrocytes resistance to an acid hemolytic, an intensified uptake of glucose, and increased activity of glycolytic enzymes (hexokinase, aldolase, phosphohexoisomerase), accumulation of lactate, along with changes in the redox enzymes activity, suppression of the catalase and intensification of peroxidase activity. Vitamin A 30-37 catalase Rattus norvegicus 411-419 10939630-8 2000 These results suggest that retinol treatment induced ornithine decarboxylase and catalase activity and increased malondialdehyde concentration. Vitamin A 27-34 catalase Rattus norvegicus 81-89 21092734-8 2011 Vitamin A supplementation during pregnancy and nursing increased superoxide dismutase/catalase (SOD/CAT) ratio and oxidative damage in maternal and offspring striatum and hippocampus. Vitamin A 0-9 catalase Rattus norvegicus 100-103 22806338-6 2012 Vitamin A at 1,500 IUkg(-1) day(-1) decreased exploratory behavior and decreased total hippocampal thiol content in sham-operated rats, increased hippocampal SOD/CAT ratio and decreased total antioxidant potential in the hippocampus of both sham and OVX groups, and increased cortical TBARS levels in OVX rats. Vitamin A 0-9 catalase Rattus norvegicus 162-165 19842843-0 2009 Vitamin A supplementation induces oxidative stress and decreases the immunocontent of catalase and superoxide dismutase in rat lungs. Vitamin A 0-9 catalase Rattus norvegicus 86-94 20041796-4 2009 Vitamin A supplementation at either therapeutic (1,000 and 2,500 IU/kg) or excessive (4,500 and 9,000 IU/kg) doses for 3, 7, or 28 days induced lipid peroxidation, protein carbonylation, and oxidation of protein thiol groups, as well as change in catalase and superoxide dismutase activity. Vitamin A 0-9 catalase Rattus norvegicus 247-255 19842843-4 2009 Vitamin A supplementation either at therapeutic (1000 and 2500 IU/kg) or excessive (4500 and 9000 IU/kg) doses for 28 days induced lipid peroxidation, protein carbonylation, and oxidation of protein thiol groups, as well as change in catalase (EC 1.11.1.6; CAT) and superoxide dismutase (EC 1.15.1.1, SOD) activities and immunocontents. Vitamin A 0-9 catalase Rattus norvegicus 234-242 16036340-8 2005 Lower than control activities of CuZnSOD, GPx, and CAT were observed in aorta of the vitamin A- deficient group. Vitamin A 85-94 catalase Rattus norvegicus 51-54 17712631-5 2008 Vitamin A supplementation induced lipid peroxidation, protein carbonylation, and oxidation of protein thiol groups, as well as change in catalase (CAT), superoxide dismutase (SOD), and glutathione peroxidase (GPx) activity. Vitamin A 0-9 catalase Rattus norvegicus 137-145 17712631-5 2008 Vitamin A supplementation induced lipid peroxidation, protein carbonylation, and oxidation of protein thiol groups, as well as change in catalase (CAT), superoxide dismutase (SOD), and glutathione peroxidase (GPx) activity. Vitamin A 0-9 catalase Rattus norvegicus 147-150 17673185-7 2007 Acutely, both therapeutic and excessive vitamin A doses induced a 1.8- to 2.2-fold decrease of catalase (CAT) activity, as determined through the rate of decrease of hydrogen peroxide (H(2)O(2)). Vitamin A 40-49 catalase Rattus norvegicus 105-108 17673185-11 2007 Furthermore, our results show that vitamin A supplementation impaired the SOD/CAT ratio. Vitamin A 35-44 catalase Rattus norvegicus 78-81 19217259-7 2009 In the vitamin A-deficient groups, liver carbonyl protein increased, whereas catalase and glutathione S-transferase activities decreased, suggesting that vitamin A protects the liver against protein oxidation. Vitamin A 7-16 catalase Rattus norvegicus 77-85 19217259-7 2009 In the vitamin A-deficient groups, liver carbonyl protein increased, whereas catalase and glutathione S-transferase activities decreased, suggesting that vitamin A protects the liver against protein oxidation. Vitamin A 154-163 catalase Rattus norvegicus 77-85 17673185-7 2007 Acutely, both therapeutic and excessive vitamin A doses induced a 1.8- to 2.2-fold decrease of catalase (CAT) activity, as determined through the rate of decrease of hydrogen peroxide (H(2)O(2)). Vitamin A 40-49 catalase Rattus norvegicus 95-103