PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29756671-5	2018	Interestingly, 6-MSITC inhibited the cell proliferation in both types of cells with similar IC50 value although a light increase in the phosphorylation and accumulation of P53 protein was observed in HCT116 p53+/+ cells at 24 h after treatment.	6-(Methylsulfinyl)hexyl isothiocyanate	15-22	tumor protein p53	Homo sapiens	207-210	
30730256-5	2019	Signaling data revealed that the activation of ERK1/2, rather than p53, is recruited for 6-MSITC-induced apoptosis.	6-(Methylsulfinyl)hexyl isothiocyanate	89-96	tumor protein p53	Homo sapiens	67-70	
29756671-5	2018	Interestingly, 6-MSITC inhibited the cell proliferation in both types of cells with similar IC50 value although a light increase in the phosphorylation and accumulation of P53 protein was observed in HCT116 p53+/+ cells at 24 h after treatment.	6-(Methylsulfinyl)hexyl isothiocyanate	15-22	tumor protein p53	Homo sapiens	172-175	
29756671-6	2018	In addition, 6-MSITC increased the ratio of proapoptotic cells in both types of cells with the same fashion in a p53-independent manner.	6-(Methylsulfinyl)hexyl isothiocyanate	13-20	tumor protein p53	Homo sapiens	113-116	
29756671-9	2018	These findings suggest that 6-MSITC might be a potential agent for colon cancer chemoprevention although with p53 mutation.	6-(Methylsulfinyl)hexyl isothiocyanate	28-35	tumor protein p53	Homo sapiens	110-113