PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19467701-4	2009	BisGMA (0.1 mm) also stimulated ERK phosphorylation, PGE(2) production, COX-2 mRNA and protein expression as well as ROS production (as indicated by an increase in cellular DCF fluorescence) in dental pulp cells.	Bisphenol A-Glycidyl Methacrylate	0-6	mitochondrially encoded cytochrome c oxidase II	Homo sapiens	72-77	
19467701-5	2009	Catalase (500 and 1000 U/ml) and U0126 (10 and 20 microm, a MEK inhibitor) effectively prevented the BisGMA-induced ERK activation, PGE(2) production and COX-2 expression.	Bisphenol A-Glycidyl Methacrylate	101-107	mitochondrially encoded cytochrome c oxidase II	Homo sapiens	154-159	
19467701-7	2009	These results suggest that BisGMA released from composite resin may potentially affect the vitality of dental pulp and induce pulpal inflammation via stimulation of ROS production, MEK/ERK1/2 activation and subsequent COX-2 gene expression and PGE(2) production.	Bisphenol A-Glycidyl Methacrylate	27-33	mitochondrially encoded cytochrome c oxidase II	Homo sapiens	218-223