PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26254990-9	2015	In summary, Ponatinib reversed the EMT, inhibited the apoptosis of AT I, as well as HLF-1 proliferation and prevented pulmonary fibrosis by suppressing the TGF-beta1/Smad3 pathway.	ponatinib	12-21	transforming growth factor beta 1	Homo sapiens	156-165	
26254990-0	2015	Ponatinib ameliorates pulmonary fibrosis by suppressing TGF-beta1/Smad3 pathway.	ponatinib	0-9	transforming growth factor beta 1	Homo sapiens	56-65	
26254990-4	2015	In this study, the effects of Ponatinib on TGF-beta1-mediated epithelial-mesenchymal transition (EMT) in A549 cells, on the proliferation of human lung fibroblasts (HLF-1), on the apoptosis of human type I alveolar epithelial cells (AT I) in vitro, and on bleomycin (BLM)-induced pulmonary fibrosis was investigated in vivo.	ponatinib	30-39	transforming growth factor beta 1	Homo sapiens	43-52