PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9704053-4	1997	IL-1-induced lung leak was decreased by treatment with superoxide dismutase (SOD), dimethylsulfoxide (DMSO), supercritical fluid-aerosolized vitamin E, interleukin-1-receptor antagonist (IL-1ra), or liposome-associated PGE1 (Lip-PGE1).	Dimethyl Sulfoxide	83-100	interleukin 1 alpha	Homo sapiens	0-4	
20021188-3	2003	We have shown that the allergens nickel, chromium, isoeugenol, and dinitrofluoro benzene induce the secretion of IL-1 betaat levels that are two- to threefold higher than those of controls and that the nonallergens and irritants sodium dodecyl sulfate, Tween-20, acetic acid, sodium hydroxide, and dimethyl sulfoxide fail to induce such a response.	Dimethyl Sulfoxide	298-316	interleukin 1 alpha	Homo sapiens	113-117	
9704053-4	1997	IL-1-induced lung leak was decreased by treatment with superoxide dismutase (SOD), dimethylsulfoxide (DMSO), supercritical fluid-aerosolized vitamin E, interleukin-1-receptor antagonist (IL-1ra), or liposome-associated PGE1 (Lip-PGE1).	Dimethyl Sulfoxide	102-106	interleukin 1 alpha	Homo sapiens	0-4	
34727650-6	2021	Results: Compared with the control group and DMSO group, the expression levels of IL-1beta, IL-6, TNF-alpha in alveolar macrophages decreased significantly in the ATL-I group (P<0.05) , and the expression levels of p-NF-kappaB, the ratio of LC3-II/LC3-I also decreased significantly in the ATL-I group (P<0.05) .	Dimethyl Sulfoxide	45-49	interleukin 1 alpha	Homo sapiens	82-90	
21878375-5	2011	DMSO at 0.5% decreased significantly mRNA levels of 14 proteins involved in the inflammatory response (including IL-6, IL-1alpha, IL-1beta, and COX-2).	Dimethyl Sulfoxide	0-4	interleukin 1 alpha	Homo sapiens	119-128