PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21161674-5	2011	Pyrrolidine dithiocarbamate (PDTC) treatment inhibited NF-kappaB activation and Bcl-2 expression and committed resveratrol-treated cells to apoptosis at the 8-h time point without the step of neuron-oriented differentiation.	pyrrolidine dithiocarbamic acid	0-27	BCL2 apoptosis regulator	Homo sapiens	80-85	
21161674-5	2011	Pyrrolidine dithiocarbamate (PDTC) treatment inhibited NF-kappaB activation and Bcl-2 expression and committed resveratrol-treated cells to apoptosis at the 8-h time point without the step of neuron-oriented differentiation.	pyrrolidine dithiocarbamic acid	29-33	BCL2 apoptosis regulator	Homo sapiens	80-85	
10777712-7	2000	The antioxidant and GSH precursor N-acetyl-l-cysteine favored Bcl-2 at the expense of Bax/p53, whereas pyrrolidine dithiocarbamate induced Bax against Bcl-2, with mild effect on p53.	pyrrolidine dithiocarbamic acid	103-130	BCL2 apoptosis regulator	Homo sapiens	151-156	
10807870-11	2000	The importance of NF-kappaB in endothelial apoptosis was confirmed by the observation that pyrrolidine dithiocarbamate, a potent NF-kappaB inhibitor, prevented endothelial apoptosis, caspase 3-like activity, and bcl-2 downregulation induced by hypoxia.	pyrrolidine dithiocarbamic acid	91-118	BCL2 apoptosis regulator	Homo sapiens	212-217	
15298726-4	2004	After treatment with these agents, the phosphorylation of protein kinase A, Bcl-2 (Thr56) and Bad (Ser155) was increased, especially in antioxidant (N-acetylcysteine and pyrrolidine dithiocarbamate)-pretreated control cells, but the phosphorylation levels were very low in the antisense-transfected cells.	pyrrolidine dithiocarbamic acid	170-197	BCL2 apoptosis regulator	Homo sapiens	76-81	
12482581-8	2002	Bcl-2 phosphorylation was increased by hydroxytyrosol and pyrrolidine dithiocarbamate and not by resveratrol.	pyrrolidine dithiocarbamic acid	58-85	BCL2 apoptosis regulator	Homo sapiens	0-5