PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 30763608-7 2019 The parasympatholytic compound scopolamine had the strongest relaxing effect (EC50 = 0.05 muM) followed by atropine (EC50 = 0.07 muM). Atropine 107-115 latexin Homo sapiens 129-132 26928772-1 2016 The contractile effect of AITC (300 muM) on human jejunal longitudinal strips was inhibited by the TRPA1 antagonist HC 030031 and atropine or scopolamine, but was insensitive to tetrodotoxin, purinoceptor antagonists or capsaicin desensitization. Atropine 130-138 latexin Homo sapiens 36-39 27108935-6 2016 The related molecule, atropine, similarly inhibited 5-HT evoked responses in oocytes with an IC50 of 1.74 muM, and competed with G-FL with a Ki of 7.94 muM. Atropine 22-30 latexin Homo sapiens 106-109 27108935-6 2016 The related molecule, atropine, similarly inhibited 5-HT evoked responses in oocytes with an IC50 of 1.74 muM, and competed with G-FL with a Ki of 7.94 muM. Atropine 22-30 latexin Homo sapiens 152-155 26862290-8 2016 ACh (100 muM) induced contractions were attenuated (by 60%) by atropine. Atropine 63-71 latexin Homo sapiens 9-12 22001099-7 2011 Stretching of the tissue also caused an increase in the spontaneous contractile rate, and these responses were abolished by atropine (1 muM) and low concentrations of 4-diphenylacetoxy-N-methylpiperidine methiodide (10 nM). Atropine 124-132 latexin Homo sapiens 136-139 26485315-8 2015 Thus, a complex made at a lower loading ratio (<= 0.1) displayed greater atropine affinity (KD muM) than other complexes prepared at higher ratios (>10), which showed only mM affinity. Atropine 76-84 latexin Homo sapiens 100-103 26320635-6 2015 A very low concentration of Ru(bpy)32+, namely 9 muM, was demonstrated to be enough for achieving the sensitive detection of TPrA, nicotine and atropine. Atropine 144-152 latexin Homo sapiens 49-52 23511063-5 2013 This atropine sensitive component amounted to~95% in bladder and ~75% in ileum, and it was enhanced by distigmine in a concentration dependent manner (0.1-3 muM; ~100-600% increase in bladder and ~50-250% increase in ileum). Atropine 5-13 latexin Homo sapiens 157-160 22525378-9 2012 Also in the presence of atropine (1 muM) and TC (30 muM) a significant effect was not detected (5800+-1300 [n=10]). Atropine 24-32 latexin Homo sapiens 36-39 22336044-6 2012 Under the optimized experimental conditions, the detection limits (3s) for raceanisodamine hydrochloride and atropine sulfate were 0.11 muM, 0.09 muM, respectively. Atropine 109-125 latexin Homo sapiens 136-139 22336044-6 2012 Under the optimized experimental conditions, the detection limits (3s) for raceanisodamine hydrochloride and atropine sulfate were 0.11 muM, 0.09 muM, respectively. Atropine 109-125 latexin Homo sapiens 146-149 626919-6 1978 In the presence of the muscarinic antagonist, atropine, acetylcholine stimulated dopamine release, whereas in the presence of the nicotinic antagonists, hexamethonium (0.2 mM) or alpha-bungaro-toxin (0.188 muM), acetylcholine inhibited dopamine release. Atropine 46-54 latexin Homo sapiens 206-209 20876203-9 2010 Atropine (1 muM) reduced the evoked responses in ICC-MY, and subsequent addition of an NK1 antagonist (RP 67580, 500 nM) completely blocked the responses to stimulation, as did applying these drugs in reverse order. Atropine 0-8 latexin Homo sapiens 12-15 200297-3 1977 Atropine (0.14 muM) reduced the secretion of amylase but did not abolish the effect.5 Adenylate cyclase prepared from cat pancreas, was stimulated by the octapeptide of cholecystokinin-pancreozymin, secretin and sodium fluoride.6 Alloxanate strongly inhibited both basal and hormone-stimulated adenylate cyclase activity. Atropine 0-8 latexin Homo sapiens 15-18