PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21994270-6	2012	The response facilitation was completely or strongly blocked by atropine (At), a muscarinic ACh receptor (mAChR) antagonist, in almost all neurons (96% of cells), whereas any residual effect after At administration was fully removed by mecamylamine, a nicotinic AChR (nAChR) antagonist, suggesting a predominant role for mAChRs in this mechanism.	Atropine	64-72	cholinergic receptor nicotinic alpha 4 subunit	Homo sapiens	268-273	
21994270-6	2012	The response facilitation was completely or strongly blocked by atropine (At), a muscarinic ACh receptor (mAChR) antagonist, in almost all neurons (96% of cells), whereas any residual effect after At administration was fully removed by mecamylamine, a nicotinic AChR (nAChR) antagonist, suggesting a predominant role for mAChRs in this mechanism.	Atropine	74-76	cholinergic receptor nicotinic alpha 4 subunit	Homo sapiens	268-273	
11756485-3	2002	With ionotropic glutamate and GABA(A) transmission blocked, this stimulation evoked fast, atropine-insensitive EPSPs that were sensitive to nAChR antagonists.	Atropine	90-98	cholinergic receptor nicotinic alpha 4 subunit	Homo sapiens	140-145