PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 16678862-0 2006 Fos immunolabelling evidence for brain regions involved in the Pavlovian degraded contingency effect and in its disruption by atropine. Atropine 126-134 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 0-3 16678862-7 2006 When atropine-induced suppression of the DCE, Fos pattern was modified in the mPFC with a change in Fos immunoreactivity, but no longer associated with the DCE. Atropine 5-13 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 46-49 16678862-7 2006 When atropine-induced suppression of the DCE, Fos pattern was modified in the mPFC with a change in Fos immunoreactivity, but no longer associated with the DCE. Atropine 5-13 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 100-103 24723855-4 2014 Atropine blocked c-Fos expression in the cortex and BG, despite high c-Fos expression in the sub-cortical arousal neuronal groups and thalamus, indicating that cortical activity is required for BG activation. Atropine 0-8 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 17-22 19531280-6 2010 Here, we set out to test the actions of typical, atypical and potential antipsychotics on atropine-induced disruption of the DCE and the related mPFC Fos-immunoreactivity profile. Atropine 90-98 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 150-153 19531280-9 2010 Compared to contingent control groups, an increased level of Fos immunoreactivity within the mPFC was observed only with doses that reversed atropine-induced disruption of the DCE. Atropine 141-149 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 61-64 16678862-6 2006 Compared to the normal DCE, atropine-induced disruption of the DCE was associated with an increase of the amount of Fos immunoreactive neurons within the central nucleus of the amygdala. Atropine 28-36 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 116-119 11478926-8 2001 Addition of atropine to the dialysis medium attenuated the increase of Fos-IR and suppressed the cholinergic stimulation-induced responses in body temperature and water intake. Atropine 12-20 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 71-74 15982643-2 2005 Pretreatment with the antimuscarinic drugs scopolamine and atropine was able to greatly suppress novelty-induced Fos expression at these sites. Atropine 59-67 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 113-116 10409218-4 1999 Atropine and hexamethonium partially decreased c-fos expression (banding vs. banding + atropine/hexamethonium: 700 +/- 67% vs. 400 +/- 67%, P < 0.05). Atropine 0-8 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 47-52 11484778-7 2001 Addition of atropine, a muscarinic receptor antagonist, to the dialysis medium containing neostigmine attenuated the increase of Fos-IR and suppressed the neostigmine-induced responses in body temperature. Atropine 12-20 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 129-132 11471222-4 2001 Intracerebroventricular injection of atropine partly blocked the SFO stimulation-induced drinking behavior and the Fos protein expression in the brain, suggesting that an M-cholinergic mechanism may be involved. Atropine 37-45 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 115-118 11378256-5 2001 In somatosensory cortex, the induction of the c-fos gene by linopirdine was nearly completely blocked by atropine and scopolamine and strongly attenuated by the NMDA receptor blockers CPP and MK-801. Atropine 105-113 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 46-51 10409218-4 1999 Atropine and hexamethonium partially decreased c-fos expression (banding vs. banding + atropine/hexamethonium: 700 +/- 67% vs. 400 +/- 67%, P < 0.05). Atropine 87-95 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 47-52 7968354-4 1994 Pretreatment of animals with atropine or pirenzepine significantly reduced induction of c-fos, jun-B, krox-20 and krox-24 genes in both hippocampus and cortex. Atropine 29-37 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 88-93 7891139-11 1995 Pretreatment of the rats with chlorisondamine alone or in combination with atropine diminished the capsaicin-induced increase in c-fos, whereas atropine alone was less efficient. Atropine 75-83 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 129-134 8281324-10 1993 Atropine, a non-specific muscarinic antagonist, also induced Fos immunoreactivity in the striatum but with matrix predominance (mostly in substance P neurons), as well as in the cingulate cortex, and the olfactory tubercle. Atropine 0-8 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 61-64 8298998-2 1993 Augmentation of c-fos and NGFI-A mRNA by light is apparently associated with activation of cholinergic nicotinic and muscarinic receptors as it can be suppressed by the nicotinic antagonist mecamylamine and the muscarinic antagonist atropine. Atropine 233-241 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 16-21 8469394-6 1993 Both atropine (10 mg/kg) and pirenzepine (100 mg/kg) reduced FOS induction suggesting that a pirenzepine-sensitive muscarinic receptor was involved. Atropine 5-13 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 61-64 1450906-1 1992 The effects of intraseptal application of atropine on c-fos proto-oncogene expression related to soman treatment were studied by immunohistochemistry for c-Fos-like proteins. Atropine 42-50 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 54-59 1450906-4 1992 The intraseptal application of atropine, which prevented soman-induced convulsions, reduced or even blocked c-Fos-like protein production related to soman treatment. Atropine 31-39 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 108-113