PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17000004-3	2007	In this work, we provide evidence that the induction of HBD-2 mRNA in response to Mycobacterium bovis bacillus Calmette-Guerin (BCG) was inhibited in a dose-dependent manner by pretreatment with a cell-permeable BAPTA-AM, which chelates intracellular calcium.	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	212-220	defensin beta 4A	Homo sapiens	56-61	
14578498-6	2003	BAPTA-AM inhibited hBD-2 induction by both thapsigargin and calcium in a dose-dependent fashion.	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	0-8	defensin beta 4A	Homo sapiens	19-24	
14578498-7	2003	In addition, BAPTA-AM inhibited hBD-2 induction by a bacterial stimulant.	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	13-21	defensin beta 4A	Homo sapiens	32-37	
11298331-4	2001	Interestingly, histamine release and intracellular Ca(2+) mobilization elicited by hBD-2 and LL-37 were markedly suppressed by BAPTA-AM (an intracellular Ca(2+) chelating agent), pertussis toxin and U-73122 (a phospholipase C inhibitor).	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	127-135	defensin beta 4A	Homo sapiens	83-88