PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30547064-2	2018	Data illustrate hypoxia-induced VEGF and leptin expression in human adipocytes treated with the calcium chelator BAPTA-AM (1 microM).	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	113-121	vascular endothelial growth factor A	Homo sapiens	32-36	
17549302-3	2007	Consistent with the importance of this pathway for tissue factor induction, treatment of endothelial cells with the Ca(++) chelator BAPTA-AM, as well as the calcineurin inhibitor cyclosporin A, partially inhibited VEGF-induced tissue factor upregulation.	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	132-140	vascular endothelial growth factor A	Homo sapiens	214-218	
20621840-2	2010	We demonstrate that reduction in Ca(2+) by chelating compound BAPTA-AM or by IP(3)-endoplasmic reticulum blocker 2-APB selectively inhibited VEGF-induced activation of c-Src-PI3K-Akt but not ERK1/2 in human coronary artery endothelial cells (HCAEC).	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	62-70	vascular endothelial growth factor A	Homo sapiens	141-145	
18752579-9	2008	Furthermore, VEGF-induced secretion is largely preserved when the rise in [Ca2+]c is prevented by BAPTA-AM.	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	98-106	vascular endothelial growth factor A	Homo sapiens	13-17	
12893832-7	2003	Moreover, the MEK inhibitor PD98059 or expression of dominant negative MEK1 inhibited induction of RANK by VEGF but not the Ca2+ chelator 1,2-bis(2-aminophenoxy)ethane-N,N,N",N"-tetraacetic acid-acetoxymethyl ester (BAPTA-AM).	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	216-224	vascular endothelial growth factor A	Homo sapiens	107-111	
10358107-10	1999	In the presence of extracellular Ca2+ the membrane permeant calcium chelator BAPTA-AM stimulated the production of EPO (P < 0.05) but not of VEGF while EGTA-AM, a closely related agent, affected neither EPO nor VEGF formation under these conditions.	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	77-85	vascular endothelial growth factor A	Homo sapiens	214-218	
8954965-4	1996	Moreover, the cells treated with BAPTA-AM prior to hypoxia also show a dramatic decrease in the VPF/VEGF message level, which suggests an important role of intracellular Ca2+ in this signaling pathway.	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	33-41	vascular endothelial growth factor A	Homo sapiens	96-99	
8954965-4	1996	Moreover, the cells treated with BAPTA-AM prior to hypoxia also show a dramatic decrease in the VPF/VEGF message level, which suggests an important role of intracellular Ca2+ in this signaling pathway.	1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester	33-41	vascular endothelial growth factor A	Homo sapiens	100-104