PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26683082-0	2016	Fasudil, a Rho kinase inhibitor, promotes the autophagic degradation of A53T alpha-synuclein by activating the JNK 1/Bcl-2/beclin 1 pathway.	fasudil	0-7	mitogen-activated protein kinase 8	Homo sapiens	111-116	
26683082-7	2016	Its underlying mechanism was supported by that fasudil could increase the macroautophagy activation via JNK 1 and Bcl-2 phosphorylation and beclin 1/Vps34 complex formation.	fasudil	47-54	mitogen-activated protein kinase 8	Homo sapiens	104-109	
19052484-8	2009	In addition, fasudil significantly decreased 5-HT-induced JNK activation, ERK translocation to the nucleus and subsequent c-fos and c-jun expression.	fasudil	13-20	mitogen-activated protein kinase 8	Homo sapiens	58-61	
19052484-10	2009	Fasudil effectively suppressed 5-HT-induced PASMC proliferation and cell-cycle progression, which was associated with inhibition of JNK activation, ERK translocation to nucleus and subsequent c-fos and c-jun expression.	fasudil	0-7	mitogen-activated protein kinase 8	Homo sapiens	132-135	
15998434-9	2005	Fasudil inhibited phosphorylation of ERK1/2, JNK, and p38.	fasudil	0-7	mitogen-activated protein kinase 8	Homo sapiens	45-48