PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20051247-11	2010	Furthermore, CsA treatment prevented early Ca(2+) overload related to decreased NCX function.	Cyclosporine	13-16	solute carrier family 8 member A1	Rattus norvegicus	80-83	
23224887-2	2013	We have shown that cyclosporin A (CsA) treatment of NCX1-, NCX2-, or NCX3-transfected HEK 293 cells and non-transfected H9c2, L6, and aortic smooth muscle cells, which express NCX1 protein naturally, reduces NCX surface expression and transport activity but has no impact on total cell NCX protein.	Cyclosporine	34-37	solute carrier family 8 member A1	Rattus norvegicus	176-180	
23224887-2	2013	We have shown that cyclosporin A (CsA) treatment of NCX1-, NCX2-, or NCX3-transfected HEK 293 cells and non-transfected H9c2, L6, and aortic smooth muscle cells, which express NCX1 protein naturally, reduces NCX surface expression and transport activity but has no impact on total cell NCX protein.	Cyclosporine	34-37	solute carrier family 8 member A1	Rattus norvegicus	52-55	
23224887-2	2013	We have shown that cyclosporin A (CsA) treatment of NCX1-, NCX2-, or NCX3-transfected HEK 293 cells and non-transfected H9c2, L6, and aortic smooth muscle cells, which express NCX1 protein naturally, reduces NCX surface expression and transport activity but has no impact on total cell NCX protein.	Cyclosporine	34-37	solute carrier family 8 member A1	Rattus norvegicus	59-62	
11700317-1	2002	Cyclosporin A (CsA) treatment of HEK 293 cells expressing the rat heart RHE-1 (NCX1.1, EMBL accession number ) or the rat brain RBE-2 (NCX1.5, GenBank(TM) accession number ) Na(+)-Ca(2+) exchanger inhibited their transport activity in a concentration-dependent manner.	Cyclosporine	0-13	solute carrier family 8 member A1	Rattus norvegicus	135-139