PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34570349-5	2022	To assess whether stroke outcomes could be improved by elevating astrocytic miR-20a-3p, we created a tetracycline (Tet)-induced recombinant adeno-associated virus (rAAV) construct where miR-20a-3p was located downstream a glial fibrillary acidic protein promoter.	Tetracycline	101-113	glial fibrillary acidic protein	Homo sapiens	222-253	
10740230-3	2000	This experimental system creates an "on/off" situation in which GFAP expression is suppressed by tetracycline.	Tetracycline	97-109	glial fibrillary acidic protein	Homo sapiens	64-68	
34570349-5	2022	To assess whether stroke outcomes could be improved by elevating astrocytic miR-20a-3p, we created a tetracycline (Tet)-induced recombinant adeno-associated virus (rAAV) construct where miR-20a-3p was located downstream a glial fibrillary acidic protein promoter.	Tetracycline	115-118	glial fibrillary acidic protein	Homo sapiens	222-253	
11124058-2	2000	To achieve combined cell-type-specific transcriptional targeting and inducible expression, we have engineered the expression of the tetracycline-dependent transcriptional elements (1) to be under the transcriptional control of either the astrocyte-specific, glial fibrillary acidic protein (GFAP) (2) or the neuronal specific enolase (NSE) promoter (3) within a dual adenoviral vector system.	Tetracycline	132-144	glial fibrillary acidic protein	Homo sapiens	258-289	
20824710-7	2011	Reduction of Sox2 expression by using siRNA against Sox2 or by overexpressing Sox21 using a tetracycline-regulated expression system (Tet-on) caused decreased GFAP expression and a reduction in cell number due to induction of apoptosis.	Tetracycline	92-104	glial fibrillary acidic protein	Homo sapiens	159-163	
11124058-2	2000	To achieve combined cell-type-specific transcriptional targeting and inducible expression, we have engineered the expression of the tetracycline-dependent transcriptional elements (1) to be under the transcriptional control of either the astrocyte-specific, glial fibrillary acidic protein (GFAP) (2) or the neuronal specific enolase (NSE) promoter (3) within a dual adenoviral vector system.	Tetracycline	132-144	glial fibrillary acidic protein	Homo sapiens	291-295	
11124058-4	2000	We demonstrate that the GFAP promoter is able to restrict tetracycline-dependent transgene expression to glial cells in cell lines, primary cultures, and in the CNS in vivo.	Tetracycline	58-70	glial fibrillary acidic protein	Homo sapiens	24-28