PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16200064-5	2005	Most notably, by tightly regulating Trp53 knock-down using tetracycline-based systems, we show that cultured mouse fibroblasts can be switched between proliferative and senescent states and that tumors induced by Trp53 suppression and cooperating oncogenes regress upon re-expression of Trp53.	Tetracycline	59-71	transformation related protein 53	Mus musculus	36-41	
17572676-4	2007	Transgenic mice harboring a tet-responsive RNA polymerase II promoter driving a microRNA-based short hairpin RNA targeting the tumor suppressor Trp53 reversibly express short hairpin RNA when crossed with existing mouse strains expressing general or tissue-specific "tet-on" or "tet-off" transactivators.	Tetracycline	28-31	transformation related protein 53	Mus musculus	144-149	
17572676-4	2007	Transgenic mice harboring a tet-responsive RNA polymerase II promoter driving a microRNA-based short hairpin RNA targeting the tumor suppressor Trp53 reversibly express short hairpin RNA when crossed with existing mouse strains expressing general or tissue-specific "tet-on" or "tet-off" transactivators.	Tetracycline	267-270	transformation related protein 53	Mus musculus	144-149	
17572676-4	2007	Transgenic mice harboring a tet-responsive RNA polymerase II promoter driving a microRNA-based short hairpin RNA targeting the tumor suppressor Trp53 reversibly express short hairpin RNA when crossed with existing mouse strains expressing general or tissue-specific "tet-on" or "tet-off" transactivators.	Tetracycline	267-270	transformation related protein 53	Mus musculus	144-149	
16200064-5	2005	Most notably, by tightly regulating Trp53 knock-down using tetracycline-based systems, we show that cultured mouse fibroblasts can be switched between proliferative and senescent states and that tumors induced by Trp53 suppression and cooperating oncogenes regress upon re-expression of Trp53.	Tetracycline	59-71	transformation related protein 53	Mus musculus	213-218	
16200064-5	2005	Most notably, by tightly regulating Trp53 knock-down using tetracycline-based systems, we show that cultured mouse fibroblasts can be switched between proliferative and senescent states and that tumors induced by Trp53 suppression and cooperating oncogenes regress upon re-expression of Trp53.	Tetracycline	59-71	transformation related protein 53	Mus musculus	213-218