PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30995417-2	2019	In this study, we utilized the tetracycline-regulatable Tet-On 3G system to control the expression of c-Jun, which is a common regulator of multiple NTFs in Schwann cells (SCs).	Tetracycline	31-43	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	102-107	
11108661-3	2000	When a tetracycline-regulatable dominant-negative c-jun (TAM67, having a truncated transactivation domain) was expressed in tumorigenic human keratinocytes, AP-1- and NFkappaB- but not p53-dependent reporter activity was inhibited by 40-60%.	Tetracycline	7-19	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	50-55	
11108661-3	2000	When a tetracycline-regulatable dominant-negative c-jun (TAM67, having a truncated transactivation domain) was expressed in tumorigenic human keratinocytes, AP-1- and NFkappaB- but not p53-dependent reporter activity was inhibited by 40-60%.	Tetracycline	7-19	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	157-161	
10473668-2	1999	To extend our understanding of the role of c-Jun in EC physiology, we have directed overexpression of c-Jun in human umbilical vein ECs by using a tetracycline-regulated adenoviral expression system.	Tetracycline	147-159	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	102-107	
10473668-5	1999	Tetracycline can effectively shut off c-Jun overexpression and prevent EC apoptosis.	Tetracycline	0-12	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	38-43