PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30405800-0	2018	Low dose dinaciclib enhances doxorubicin-induced senescence in myeloma RPMI8226 cells by transformation of the p21 and p16 pathways.	dinaciclib	9-19	H3 histone pseudogene 16	Homo sapiens	111-114	
30405800-8	2018	Notably, the combination of dinaciclib and DOX inhibited cell growth and promoted senescence by transforming the suppressive effects of the ATM/Chk2/p53/p21 signaling pathway and enhancing the p16 signaling pathway.	dinaciclib	28-38	H3 histone pseudogene 16	Homo sapiens	153-156	
30405800-9	2018	Thus, low-dose dinaciclib enhanced anti-MM effects mediated by DOX via transformation of p21-p16 signaling pathways, leading to accelerated senescence, but not apoptosis.	dinaciclib	15-25	H3 histone pseudogene 16	Homo sapiens	89-92