PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29507054-7	2018	Notably, we found that MITF expression correlates with CDK2 upregulation in patients; thus, dinaciclib would warrant consideration for treatment of patients unresponsive to BRAF-MEK and/or Hsp90 inhibitors and/or harboring MITF amplification/overexpression.	dinaciclib	92-102	B-Raf proto-oncogene, serine/threonine kinase	Homo sapiens	173-177	
33030957-4	2021	Inhibition of MEK/ERK by cobimetinib or CDK inhibitor dinaciclib was shown to attenuate mutant BRAF-induced PD-L1 coincident with reduced c-JUN and YAP expression whose combined knockdown reduced PD-L1.	dinaciclib	54-64	B-Raf proto-oncogene, serine/threonine kinase	Homo sapiens	95-99	
31744894-5	2020	Contrary to dinaciclib, the inhibitors of BRAF / MEK / CDK4/6 induced apoptosis dominantly through a BAX-dependent mechanism.	dinaciclib	12-22	B-Raf proto-oncogene, serine/threonine kinase	Homo sapiens	42-46