PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28093219-11	2017	Imipramine decreased c-Fos expression in the basolateral amygdala and CA1 and CA3 divisions of the hippocampus.	Imipramine	0-10	carbonic anhydrase 3	Rattus norvegicus	78-81	
1839498-4	1991	The long-term treatments with imipramine and ECS, but not with paroxetine, increased the responsiveness of postsynaptic CA3 hippocampus pyramidal neurons to the microiontophoretic application of 5-HT and to that of the selective 5-HT1A receptor ligand 8-OH-DPAT.	Imipramine	30-40	carbonic anhydrase 3	Rattus norvegicus	120-123	
28866130-12	2018	Imipramine decreased c-Fos in the basolateral amygdala and hippocampus (CA1 and CA3), but increased c-Fos in the central amygdala.	Imipramine	0-10	carbonic anhydrase 3	Rattus norvegicus	80-83	
20572198-5	2011	Subchronic treatment with imipramine showed a tendency (although not statistically significant) to decrease the LH-induced increment of activated astrocytes in the CA3 region and dentate gyrus.	Imipramine	26-36	carbonic anhydrase 3	Rattus norvegicus	164-167	
20572198-7	2011	However, the antidepressant-like effects of imipramine in the LH paradigm were blocked when fluorocitrate (a reversible inhibitor of astrocyte function) was injected into the dentate gyrus or CA3 region.	Imipramine	44-54	carbonic anhydrase 3	Rattus norvegicus	192-195	
17950272-8	2008	In the CA3 region a reduction of GAP-43 mRNA was observed after treatment with imipramine (21 days) and fluoxetine (7 and 14 days).	Imipramine	79-89	carbonic anhydrase 3	Rattus norvegicus	7-10