PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 32934732-11 2020 Further experiments revealed that miR-101 was a target of MALAT1 and that the miR-101 inhibitor abolished the effect of MALAT1 on OSCC cell proliferation and invasion. mir-101 34-41 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 58-64 32934732-11 2020 Further experiments revealed that miR-101 was a target of MALAT1 and that the miR-101 inhibitor abolished the effect of MALAT1 on OSCC cell proliferation and invasion. mir-101 34-41 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 120-126 32934732-11 2020 Further experiments revealed that miR-101 was a target of MALAT1 and that the miR-101 inhibitor abolished the effect of MALAT1 on OSCC cell proliferation and invasion. mir-101 78-85 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 120-126 31372165-0 2019 Long non-coding RNA Malat1 activated autophagy, hence promoting cell proliferation and inhibiting apoptosis by sponging miR-101 in colorectal cancer. mir-101 120-127 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 20-26 31372165-10 2019 In addition, Malat1 was found to act as an endogenous sponge by directly binding to miR-101 to reduce miR-101. mir-101 84-91 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 13-19 31372165-10 2019 In addition, Malat1 was found to act as an endogenous sponge by directly binding to miR-101 to reduce miR-101. mir-101 102-109 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 13-19 31372165-11 2019 Furthermore, the suppressive effects of miR-101 on the autophagy, proliferation, and apoptosis of CRC were abolished by Malat1. mir-101 40-47 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 120-126 31372165-12 2019 Conclusion: Long non-coding RNA Malat1 activated autophagy and promoted cell proliferation, yet inhibited apoptosis by sponging miR-101 in colorectal cancer cells. mir-101 128-135 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 32-38 28834690-0 2017 Malat1 activates autophagy and promotes cell proliferation by sponging miR-101 and upregulating STMN1, RAB5A and ATG4D expression in glioma. mir-101 71-78 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 0-6 29479863-8 2018 Importantly, we demonstrate that MALAT1 promoted the chemoresistance through suppressing miR-101 signaling pathway via directly binding it in GBM cells. mir-101 89-96 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 33-39 29479863-9 2018 In conclusion, our study indicates that knockdown of MALAT1 reverses chemoresistance to TMZ via promoting miR-101 regulatory network in GBM and thus offers a novel prognostic marker and potential target for GBM TMZ-based chemotherapy. mir-101 106-113 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 53-59 28834690-9 2017 We found that Malat1 served as an endogenous sponge to reduce miR-101 expression by directly binding to miR-101. mir-101 62-69 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 14-20 28834690-9 2017 We found that Malat1 served as an endogenous sponge to reduce miR-101 expression by directly binding to miR-101. mir-101 104-111 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 14-20 28834690-10 2017 Moreover, Malat1 abolished the suppression effects of miR-101 on glioma cell autophagy and proliferation, which involved in upregulating the expression of miR-101 targets STMN1, RAB5A and ATG4D. mir-101 54-61 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 10-16 28834690-10 2017 Moreover, Malat1 abolished the suppression effects of miR-101 on glioma cell autophagy and proliferation, which involved in upregulating the expression of miR-101 targets STMN1, RAB5A and ATG4D. mir-101 155-162 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 10-16 28834690-11 2017 Overall, our study elucidated a novel Malat1-miR-101-STMN1/RAB5A/ATG4D regulatory network that Malat1 activates autophagy and promotes cell proliferation by sponging miR-101 and upregulating STMN1, RAB5A and ATG4D expression in glioma cells. mir-101 45-52 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 38-44 28834690-11 2017 Overall, our study elucidated a novel Malat1-miR-101-STMN1/RAB5A/ATG4D regulatory network that Malat1 activates autophagy and promotes cell proliferation by sponging miR-101 and upregulating STMN1, RAB5A and ATG4D expression in glioma cells. mir-101 45-52 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 95-101 28834690-11 2017 Overall, our study elucidated a novel Malat1-miR-101-STMN1/RAB5A/ATG4D regulatory network that Malat1 activates autophagy and promotes cell proliferation by sponging miR-101 and upregulating STMN1, RAB5A and ATG4D expression in glioma cells. mir-101 166-173 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 38-44 28834690-11 2017 Overall, our study elucidated a novel Malat1-miR-101-STMN1/RAB5A/ATG4D regulatory network that Malat1 activates autophagy and promotes cell proliferation by sponging miR-101 and upregulating STMN1, RAB5A and ATG4D expression in glioma cells. mir-101 166-173 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 95-101 29212230-7 2017 Further assays indicated that MALAT1 acted as a competing endogenous RNA to upregulate SOX9 expression by sponging miR-101 in DDP-resistant cancer cells, through Wnt signaling pathway. mir-101 115-122 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 30-36 28551849-0 2017 Long non-coding RNA MALAT1 promotes proliferation and suppresses apoptosis of glioma cells through derepressing Rap1B by sponging miR-101. mir-101 130-137 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 20-26 28551849-9 2017 Moreover, MALAT1 promoted the Rap1B expression by sponging miR-101 in U251 and U87 cells. mir-101 59-66 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 10-16 28551849-10 2017 Furthermore, miR-101 downregulation or Rap1B overexpression reversed the proliferation inhibitory and apoptosis induction of glioma cell lines caused by MALAT1 knockdown. mir-101 13-20 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 153-159 28551849-11 2017 Taken together, MALAT1 promotes proliferation and suppresses apoptosis of glioma cells through derepressing Rap1B by sponging miR-101. mir-101 126-133 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 16-22