PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34457117-0	2021	Salidroside Suppresses the Proliferation and Migration of Human Lung Cancer Cells through AMPK-Dependent NLRP3 Inflammasome Regulation.	rhodioloside	0-11	NLR family pyrin domain containing 3	Homo sapiens	105-110	
34457117-3	2021	It has been shown that SAL improves metabolic inflammation in diabetic rodents through AMP-activated protein kinase- (AMPK-) dependent inhibition of the NLRP3 inflammasome.	rhodioloside	23-26	NLR family pyrin domain containing 3	Homo sapiens	153-158	
34457117-4	2021	However, whether the NLRP3 inflammasome is regulated by SAL in NSCLC cells and how its underlying mechanism(s) can be determined require clarification.	rhodioloside	56-59	NLR family pyrin domain containing 3	Homo sapiens	21-26	
34457117-7	2021	Moreover, SAL protected A549 cells against LPS-induced AMPK inhibition, ROS production, and NLRP3 inflammasome activation.	rhodioloside	10-13	NLR family pyrin domain containing 3	Homo sapiens	92-97	
34457117-9	2021	In summary, these results indicate that SAL suppresses the proliferation and migration of human lung cancer cells through AMPK-dependent NLRP3 inflammasome regulation.	rhodioloside	40-43	NLR family pyrin domain containing 3	Homo sapiens	137-142	
34011327-0	2021	Salidroside protects endothelial cells against LPS-induced inflammatory injury by inhibiting NLRP3 and enhancing autophagy.	rhodioloside	0-11	NLR family pyrin domain containing 3	Homo sapiens	93-98	
34011327-12	2021	SAL also attenuated LPS-induced activation of NLRP3 inflammasome, reduced the protein expression of NLRP3-related proteins, including ASC and caspase-1.	rhodioloside	0-3	NLR family pyrin domain containing 3	Homo sapiens	46-51	
34011327-12	2021	SAL also attenuated LPS-induced activation of NLRP3 inflammasome, reduced the protein expression of NLRP3-related proteins, including ASC and caspase-1.	rhodioloside	0-3	NLR family pyrin domain containing 3	Homo sapiens	100-105	
34011327-13	2021	Autophagy inhibition by 3-MA markedly reversed SAL-modulated changes in cell viability and NLRP3 expression in LPS-stimulated HUVECs.	rhodioloside	47-50	NLR family pyrin domain containing 3	Homo sapiens	91-96	
34011327-14	2021	CONCLUSION: SAL protects endothelial cells against LPS-induced injury through inhibition of NLRP3 pathways and enhancing autophagy.	rhodioloside	12-15	NLR family pyrin domain containing 3	Homo sapiens	92-97	
32076940-9	2020	Our data indicate that SAL inhibit NLRP3-related pyroptosis, which might be the underlying mechanism of SAL anti-inflammatory in atherosclerosis.	rhodioloside	23-26	NLR family pyrin domain containing 3	Homo sapiens	35-40	
32076940-9	2020	Our data indicate that SAL inhibit NLRP3-related pyroptosis, which might be the underlying mechanism of SAL anti-inflammatory in atherosclerosis.	rhodioloside	104-107	NLR family pyrin domain containing 3	Homo sapiens	35-40	
31747547-0	2020	Salidroside ameliorates endothelial inflammation and oxidative stress by regulating the AMPK/NF-kappaB/NLRP3 signaling pathway in AGEs-induced HUVECs.	rhodioloside	0-11	NLR family pyrin domain containing 3	Homo sapiens	103-108	
31747547-10	2020	Importantly, salidroside alleviated endothelial inflammation and oxidative stress by activating AMPK phosphorylation and inhibiting NF-kB p65 and NLRP3 inflammasome activation.	rhodioloside	13-24	NLR family pyrin domain containing 3	Homo sapiens	146-151	
31747547-13	2020	Our findings suggest that salidroside ameliorates AGEs-induced endothelial inflammation and oxidative stress, partially via the AMPK/NF-kappaB/NLRP3 signaling pathway.	rhodioloside	26-37	NLR family pyrin domain containing 3	Homo sapiens	143-148	
29031534-13	2017	The expression levels of TXNIP, NLRP3, ASC, and caspase-1 were reduced in the SAL treated cells.	rhodioloside	78-81	NLR family pyrin domain containing 3	Homo sapiens	32-37