PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32002777-3	2020	In the present study, daily doses of Sal were administered to APP/PS1 mice, a mouse model of AD, and several parameters were tested, including behavioral performance, Abeta status, levels of synapse-related proteins, and levels of PI3K/Akt targets of mTOR cell signaling pathway proteins.	rhodioloside	37-40	mechanistic target of rapamycin kinase	Mus musculus	251-255	
32002777-6	2020	The phosphatidylinositide PI3K/Akt/mTOR signaling was upregulated, which was in accordance with the above improvements from Sal treatment.	rhodioloside	124-127	mechanistic target of rapamycin kinase	Mus musculus	35-39	
27493875-0	2016	Salidroside alleviates cachexia symptoms in mouse models of cancer cachexia via activating mTOR signalling.	rhodioloside	0-11	mechanistic target of rapamycin kinase	Mus musculus	91-95	
27493875-11	2016	Further analysis demonstrated that salidroside could significantly increase expression of mTOR, p-mTOR, and MyHC in gastrocnemius muscle.	rhodioloside	35-46	mechanistic target of rapamycin kinase	Mus musculus	90-94	
27493875-11	2016	Further analysis demonstrated that salidroside could significantly increase expression of mTOR, p-mTOR, and MyHC in gastrocnemius muscle.	rhodioloside	35-46	mechanistic target of rapamycin kinase	Mus musculus	98-102	
27493875-12	2016	Also, results in vitro showed that salidroside could not only obviously increase mTOR, p-mTOR, and MyHC expression in C2C12 myotubes but also effectively rescue their down-regulation induced by tumour necrosis factor-alpha.	rhodioloside	35-46	mechanistic target of rapamycin kinase	Mus musculus	81-85	
27493875-12	2016	Also, results in vitro showed that salidroside could not only obviously increase mTOR, p-mTOR, and MyHC expression in C2C12 myotubes but also effectively rescue their down-regulation induced by tumour necrosis factor-alpha.	rhodioloside	35-46	mechanistic target of rapamycin kinase	Mus musculus	89-93	
24187831-0	2013	[Salidroside via ERK1/2 and PI3K/AKT/mTOR signal pathway induces mouse bone marrow mesenchymal stem cells differentiation into neural cells].	rhodioloside	1-12	mechanistic target of rapamycin kinase	Mus musculus	37-41	
24187831-10	2013	It points out that SD can stimulate the ERK1/2 and PI3K/AKT/mTOR signal pathway to promote BMSCs differentiation into neural cells.	rhodioloside	19-21	mechanistic target of rapamycin kinase	Mus musculus	60-64	
34181207-7	2021	The results showed that mTOR could be recruited to the mitochondria after salidroside treatment, which might be responsible for inhibiting excessive mitophagy caused by OGD.	rhodioloside	74-85	mechanistic target of rapamycin kinase	Mus musculus	24-28	
32910486-8	2020	Besides, SAL can also up-regulate the mTOR/p70S6k signaling pathway in the PA-induced GLUTag cells model.	rhodioloside	9-12	mechanistic target of rapamycin kinase	Mus musculus	38-42	
32910486-11	2020	Furthermore, the SAL has also stimulated the mTOR/p70S6k signaling pathway in PA-induced GLUTag cells model.	rhodioloside	17-20	mechanistic target of rapamycin kinase	Mus musculus	45-49