PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31704291-6	2019	Our results demonstrated that pinocembrin treatment significantly decreased sympathetic activity, augmented parasympathetic activity, improved heart rate variability (HRV), prolonged the atrial effective refractory period (ERP) and action potential duration (APD), shortened activation latency (AL), lowered the indicibility rate of AF, attenuated atrial fibrosis, and decreased concentrations of norepinephrine (NE), tumor necrosis factor-alpha (TNF-alpha), interleukin (IL)-1beta and IL-6 in the serum and the left atrial (LA).	pinocembrin	30-41	interleukin 1 beta	Rattus norvegicus	459-481