PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27696114-5	2017	PB (25 muM) pretreatment (for 4 h) downregulated the levels of Bcl-2-associated X protein (Bax), blocked the release of cytochrome c to the cytosol, and inhibited the PQ-induced activation of caspase-9 and caspase-3.	pinocembrin	0-2	caspase 3	Homo sapiens	206-215	
34336656-7	2021	PCB downregulated the expression levels of proteins involved in cell cycle progression and apoptosis, including cyclinB1, Cdc2, PARP1, Bcl-2, and survivin, and upregulated protein levels of cleaved PARP1, cleaved caspase3, cleaved caspase9, and BAX.	pinocembrin	0-3	caspase 3	Homo sapiens	213-221	
27538638-7	2016	In addition, PCB suppressed the release of cytochrome c and the cleavage of caspase-3.	pinocembrin	13-16	caspase 3	Homo sapiens	76-85	
25271424-8	2014	Pinocembrin-treatment could also reduce the number of TUNEL-positive and Caspase-3-positive neurons, and upregulate the expression of LC3B and Beclin1 in penumbra area.	pinocembrin	0-11	caspase 3	Homo sapiens	73-82	
24395092-5	2014	We found that pretreatment of SH-SY5Y cells with PB significantly reduced the MPP(+)-induced loss of cell viability, the generation of intracellular ROS, apoptotic rate, and the cleavage of caspase-3.	pinocembrin	49-51	caspase 3	Homo sapiens	190-199