PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23855447-1	2013	The aim of this study was to further explore the possible mechanisms of montelukast on oral mice ovalbumin-induced eosinophilic gastroenteritis in a mouse model.	montelukast	72-83	serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene	Mus musculus	97-106	
33262813-7	2021	Inflammation scores that were increased in the OVA mice following all challenges were indicated to be reduced by montelukast treatment.	montelukast	113-124	serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene	Mus musculus	47-50	
19133568-8	2008	RESULTS: OVA-sensitized mice developed a significant airway inflammatory response that was inhibited by prednisolone and montelukast, whilst C. militaris reduced airway inflammation less effectively.	montelukast	121-132	serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene	Mus musculus	9-12	
19133568-9	2008	Airway hyperresponsiveness to methacholine was observed in OVA-sensitized mice and was reversed by both prednisolone and montelukast.	montelukast	121-132	serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene	Mus musculus	59-62	
16387808-7	2006	In OVA-treated mice, airway eosinophil infiltration and goblet cell metaplasia were reduced by either montelukast or dexamethasone alone.	montelukast	102-113	serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene	Mus musculus	3-6